Increased lymphomagenicity and restored disease specificity of AML1 site (core) mutant SL3-3 murine leukemia virus by a second-site enhancer variant evolved in vivo. (Q35894998)

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scientific article published in October 1997

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English	Increased lymphomagenicity and restored disease specificity of AML1 site (core) mutant SL3-3 murine leukemia virus by a second-site enhancer variant evolved in vivo.	scientific article published in October 1997

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6 November 2019

Increased lymphomagenicity and restored disease specificity of AML1 site (core) mutant SL3-3 murine leukemia virus by a second-site enhancer variant evolved in vivo (English)

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6 November 2019

murine leukemia virus

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murine leukemia virus

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Steen Ethelberg

1

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6 November 2019

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Lovmand J

2

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6 November 2019

Schmidt J

3

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6 November 2019

Luz A

4

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6 November 2019

Pedersen FS

5

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6 November 2019

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1 October 1997

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6 November 2019

Journal of Virology

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6 November 2019

71

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6 November 2019

10

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6 November 2019

7273-7280

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6 November 2019

Transcriptional Silencing of Retroviral Vectors.

1 reference

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Stability of AML1 (core) site enhancer mutations in T lymphomas induced by attenuated SL3-3 murine leukemia virus mutants

1 reference

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Importance of a c-Myb binding site for lymphomagenesis by the retrovirus SL3-3.

1 reference

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Second-site proviral enhancer alterations in lymphomas induced by enhancer mutants of SL3-3 murine leukemia virus: negative effect of nuclear factor 1 binding site

1 reference

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Mapping of a major osteomagenic determinant of murine leukemia virus RFB-14 to non-long terminal repeat sequences

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E-box sequence and context-dependent TAL1/SCL modulation of basic helix-loop-helix protein-mediated transcriptional activation

1 reference

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The CBFbeta subunit is essential for CBFalpha2 (AML1) function in vivo

1 reference

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Transcriptional activation of a retrovirus enhancer by CBF (AML1) requires a second factor: evidence for cooperativity with c-Myb.

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C/EBP, c-Myb, and PU.1 cooperate to regulate the neutrophil elastase promoter

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1 reference

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Sequence tags of provirus integration sites in DNAs of tumors induced by the murine retrovirus SL3-3.

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Disruption of the Cbfa2 gene causes necrosis and hemorrhaging in the central nervous system and blocks definitive hematopoiesis

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AML1, the target of multiple chromosomal translocations in human leukemia, is essential for normal fetal liver hematopoiesis

1 reference

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Cloning and characterization of subunits of the T-cell receptor and murine leukemia virus enhancer core-binding factor

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Isolation of PEBP2 alpha B cDNA representing the mouse homolog of human acute myeloid leukemia gene, AML1.

1 reference

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Fusion between transcription factor CBF beta/PEBP2 beta and a myosin heavy chain in acute myeloid leukemia

1 reference

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11 July 2018

PEBP2/PEA2 represents a family of transcription factors homologous to the products of the Drosophila runt gene and the human AML1 gene

1 reference

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The t(8;21) translocation in acute myeloid leukemia results in production of an AML1-MTG8 fusion transcript

1 reference

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11 July 2018

Cooperative binding of Ets-1 and core binding factor to DNA

1 reference

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Lack of correlation between basal expression levels and susceptibility to transcriptional shutdown among single-gene murine leukemia virus vector proviruses

1 reference

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Long terminal repeat enhancer core sequences in proviruses adjacent to c-myc in T-cell lymphomas induced by a murine retrovirus.

1 reference

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AML1, AML2, and AML3, the human members of the runt domain gene-family: cDNA structure, expression, and chromosomal localization

1 reference

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AML1 and the 8;21 and 3;21 translocations in acute and chronic myeloid leukemia

1 reference

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c-Myb and core-binding factor/PEBP2 display functional synergy but bind independently to adjacent sites in the T-cell receptor delta enhancer

1 reference

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Transcriptional activity of core binding factor-alpha (AML1) and beta subunits on murine leukemia virus enhancer cores

1 reference

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Transactivation of the Moloney murine leukemia virus and T-cell receptor beta-chain enhancers by cbf and ets requires intact binding sites for both proteins

1 reference

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Oncogenic retrovirus from spontaneous murine osteomas. I. Isolation and biological characterization

1 reference

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Determination of the leukaemogenicity of a murine retrovirus by sequences within the long terminal repeat

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Molecular cloning of a highly leukemogenic, ecotropic retrovirus from an AKR mouse

1 reference

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Tissue-specific transcription preference as a determinant of cell tropism and leukaemogenic potential of murine retroviruses

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The tandem direct repeats within the long terminal repeat of murine leukemia viruses are the primary determinant of their leukemogenic potential

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Six distinct nuclear factors interact with the 75-base-pair repeat of the Moloney murine leukemia virus enhancer

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AKR ecotropic murine leukemia virus SL3-3 forms envelope gene recombinants in vivo

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Oncogenic retrovirus from spontaneous murine osteomas. II. Molecular cloning and genomic characterization

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12 December 2020

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6 November 2019

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6 November 2019

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