Administration of Recombinant Heat Shock Protein 70 Delays Peripheral Muscle Denervation in the SOD1(G93A) Mouse Model of Amyotrophic Lateral Sclerosis (Q36151904)

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English	Administration of Recombinant Heat Shock Protein 70 Delays Peripheral Muscle Denervation in the SOD1(G93A) Mouse Model of Amyotrophic Lateral Sclerosis	scientific article

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scholarly article

1 reference

Europe PubMed Central

12 August 2017

Administration of Recombinant Heat Shock Protein 70 Delays Peripheral Muscle Denervation in the SOD1(G93A) Mouse Model of Amyotrophic Lateral Sclerosis (English)

1 reference

Europe PubMed Central

12 August 2017

amyotrophic lateral sclerosis

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author name string

David J Gifondorwa

1

1 reference

Europe PubMed Central

12 August 2017

Ramon Jimenz-Moreno

2

1 reference

Europe PubMed Central

12 August 2017

Crystal D Hayes

3

1 reference

Europe PubMed Central

12 August 2017

Hesam Rouhani

4

1 reference

Europe PubMed Central

12 August 2017

Mac B Robinson

5

1 reference

Europe PubMed Central

12 August 2017

Jane L Strupe

6

1 reference

Europe PubMed Central

12 August 2017

James Caress

7

1 reference

Europe PubMed Central

12 August 2017

Carol Milligan

8

1 reference

Europe PubMed Central

12 August 2017

publication date

1 August 2012

1 reference

Europe PubMed Central

12 August 2017

Neurology Research International

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Europe PubMed Central

12 August 2017

2012

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Europe PubMed Central

12 August 2017

170426

1 reference

Europe PubMed Central

12 August 2017

copyright license

Creative Commons Attribution 3.0 Unported

1 January 2012

1 reference

April 2022 Public Data File from Crossref

copyright status

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April 2022 Public Data File from Crossref

describes a project that uses

1 reference

Europe PubMed Central

11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC3415202/fullTextXML

based on heuristic

inferred from PubMed Central ID database lookup

Evidence of endoplasmic reticular stress in the spinal motor neurons exposed to CSF from sporadic amyotrophic lateral sclerosis patients

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The "dying-back" phenomenon of motor neurons in ALS.

1 reference

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2 October 2017

Heat shock proteins: cellular and molecular mechanisms in the central nervous system

1 reference

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Mutant SOD1 G93A microglia have an inflammatory phenotype and elevated production of MCP-1.

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Gp78, an ER associated E3, promotes SOD1 and ataxin-3 degradation.

1 reference

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A network dysfunction perspective on neurodegenerative diseases

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Fragmentation of the Golgi apparatus in neurodegenerative diseases and cell death

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Microglia as potential contributors to motor neuron injury in amyotrophic lateral sclerosis

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Treatment with arimoclomol, a coinducer of heat shock proteins, delays disease progression in ALS mice

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Axonal damage: a key predictor of outcome in human CNS diseases

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Inflammatory processes in amyotrophic lateral sclerosis

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Disease progression in amyotrophic lateral sclerosis: predictors of survival

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Axonal self-destruction and neurodegeneration

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Early and selective loss of neuromuscular synapse subtypes with low sprouting competence in motoneuron diseases

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Presymptomatic biochemical changes in hindlimb muscle of G93A human Cu/Zn superoxide dismutase 1 transgenic mouse model of amyotrophic lateral sclerosis

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Inducible isoform of HSP70 is constitutively expressed in a muscle fiber type specific pattern

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3415202

2 July 2018

A role for motoneuron subtype-selective ER stress in disease manifestations of FALS mice

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Altered sensorimotor development in a transgenic mouse model of amyotrophic lateral sclerosis

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Early vacuolization and mitochondrial damage in motor neurons of FALS mice are not associated with apoptosis or with changes in cytochrome oxidase histochemical reactivity.

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Exogenous delivery of heat shock protein 70 increases lifespan in a mouse model of amyotrophic lateral sclerosis.

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Clinical features that distinguish PLS, upper motor neuron-dominant ALS, and typical ALS.

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21 January 2018

Early detection of denervated muscle fibers in hindlimb muscles after sciatic nerve transection in wild type mice and in the G93A mouse model of amyotrophic lateral sclerosis.

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21 January 2018

Increased expression of the glial glutamate transporter EAAT2 modulates excitotoxicity and delays the onset but not the outcome of ALS in mice

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Minocycline slows disease progression in a mouse model of amyotrophic lateral sclerosis

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21 January 2018

Efficient three-drug cocktail for disease induced by mutant superoxide dismutase

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21 January 2018

Minocycline delays disease onset and mortality in a transgenic model of ALS.

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https://api.crossref.org/works/10.1155%2F2012%2F170426

21 January 2018

Minocycline inhibits cytochrome c release and delays progression of amyotrophic lateral sclerosis in mice

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https://api.crossref.org/works/10.1155%2F2012%2F170426

21 January 2018

Suppression of Alzheimer's disease-related phenotypes by expression of heat shock protein 70 in mice.

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21 January 2018

Relationship between neuropathology and disease progression in the SOD1(G93A) ALS mouse.

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23 September 2018

Preparation of PCR-quality mouse genomic DNA with hot sodium hydroxide and tris (HotSHOT).

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23 September 2018

Shifts in type I fiber proportion in rat hindlimb muscle are accompanied by changes in HSP72 content

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An intrinsic distinction in neuromuscular junction assembly and maintenance in different skeletal muscles

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https://pubmed.ncbi.nlm.nih.gov/22900172

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Amyotrophic lateral sclerosis is a distal axonopathy: evidence in mice and man

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Extracellular heat shock protein 70: a critical component for motoneuron survival

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12 December 2020

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Selective vulnerability and pruning of phasic motoneuron axons in motoneuron disease alleviated by CNTF

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https://pubmed.ncbi.nlm.nih.gov/22900172

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based on heuristic

inferred from PubMed ID database lookup

Complete dissociation of motor neuron death from motor dysfunction by Bax deletion in a mouse model of ALS

1 reference

https://pubmed.ncbi.nlm.nih.gov/22900172

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Time course of preferential motor unit loss in the SOD1 G93A mouse model of amyotrophic lateral sclerosis

1 reference

https://pubmed.ncbi.nlm.nih.gov/22900172

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Treatment with minocycline after disease onset alters astrocyte reactivity and increases microgliosis in SOD1 mutant mice

1 reference

https://pubmed.ncbi.nlm.nih.gov/22900172

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

The fragmented neuronal Golgi apparatus in amyotrophic lateral sclerosis includes the trans-Golgi-network: functional implications

1 reference

https://pubmed.ncbi.nlm.nih.gov/22900172

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Astrocytes interact intimately with degenerating motor neurons in mouse amyotrophic lateral sclerosis (ALS)

1 reference

https://pubmed.ncbi.nlm.nih.gov/22900172

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Presymptomatic motor neuron loss and reactive astrocytosis in the SOD1 mouse model of amyotrophic lateral sclerosis

1 reference

https://pubmed.ncbi.nlm.nih.gov/22900172

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Background and gender effects on survival in the TgN(SOD1-G93A)1Gur mouse model of ALS

1 reference

https://pubmed.ncbi.nlm.nih.gov/22900172

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based on heuristic

inferred from PubMed ID database lookup

A compensatory subpopulation of motor neurons in a mouse model of amyotrophic lateral sclerosis

1 reference

https://pubmed.ncbi.nlm.nih.gov/22900172

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Preferential motor unit loss in the SOD1 G93A transgenic mouse model of amyotrophic lateral sclerosis

1 reference

https://pubmed.ncbi.nlm.nih.gov/22900172

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1155/2012/170426

1 reference

Europe PubMed Central

12 August 2017

1 reference

Europe PubMed Central

12 August 2017

1 reference

Europe PubMed Central

12 August 2017

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