Apoptosis and glutathione: beyond an antioxidant. (Q37574928)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19662025%20AND%20SRC:MED&resulttype=core&format=json

4 February 2020

review article

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title

Apoptosis and glutathione: beyond an antioxidant (English)

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4 February 2020

0 references

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1

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4 February 2020

2

J A Cidlowski

2 references

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4 February 2020

21 August 2017

7 August 2009

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Cell Death & Differentiation

4 February 2020

published in

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19662025%20AND%20SRC:MED&resulttype=core&format=json

4 February 2020

volume

16

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4 February 2020

issue

10

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4 February 2020

page(s)

1303-1314

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https://scigraph.springernature.com/pub.10.1038/cdd.2009.107

4 February 2020

exact match

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In vivo reversal of glutathione deficiency and susceptibility to in vivo dexamethasone-induced apoptosis by N-acetylcysteine and L-2-oxothiazolidine-4-carboxylic acid, but not ascorbic acid, in thymocytes from gamma-glutamyltranspeptidase-deficient

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Mitochondrial glutathione protects against cell death induced by oxidative and nitrative stress in astrocytes

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Modulation of expression of rat mitochondrial 2-oxoglutarate carrier in NRK-52E cells alters mitochondrial transport and accumulation of glutathione and susceptibility to chemically induced apoptosis

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Selective mitochondrial glutathione depletion by ethanol enhances acetaminophen toxicity in rat liver

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Contribution of mitochondrial GSH transport to matrix GSH status and colonic epithelial cell apoptosis

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Endogenous glutathione adducts

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Glutathione export during apoptosis requires functional multidrug resistance-associated proteins

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Verapamil and its derivative trigger apoptosis through glutathione extrusion by multidrug resistance protein MRP1.

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Multidrug resistance-associated protein 1 as a major mediator of basal and apoptotic glutathione release

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Oatp2 mediates bidirectional organic solute transport: a role for intracellular glutathione.

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Bcl-xL overexpression attenuates glutathione depletion in FL5.12 cells following interleukin-3 withdrawal

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Redox Modulation of the Apoptogenic Activity of Thapsigargin

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Nuclear glutathione S-transferase pi prevents apoptosis by reducing the oxidative stress-induced formation of exocyclic DNA products

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GPX2, a direct target of p63, inhibits oxidative stress-induced apoptosis in a p53-dependent manner

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Increased infarct size and exacerbated apoptosis in the glutathione peroxidase-1 (Gpx-1) knockout mouse brain in response to ischemia/reperfusion injury

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Overexpression of human GPX1 modifies Bax to Bcl-2 apoptotic ratio in human endothelial cells

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Transgenic mice overexpressing glutathione peroxidase 4 are protected against oxidative stress-induced apoptosis

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Glutathione peroxidase 4 protects cortical neurons from oxidative injury and amyloid toxicity.

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Glutathione peroxidase 4 senses and translates oxidative stress into 12/15-lipoxygenase dependent- and AIF-mediated cell death

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Role for glutathione peroxidase-4 in brain development and neuronal apoptosis: specific induction of enzyme expression in reactive astrocytes following brain injury.

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Mitochondrial phospholipid hydroperoxide glutathione peroxidase inhibits the release of cytochrome c from mitochondria by suppressing the peroxidation of cardiolipin in hypoglycaemia-induced apoptosis

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Cytochrome c-mediated apoptosis in cells lacking mitochondrial DNA. Signaling pathway involving release and caspase 3 activation is conserved

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Identification of oxidant-sensitive proteins: TNF-alpha induces protein glutathiolation

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Redox amplification of apoptosis by caspase-dependent cleavage of glutaredoxin 1 and S-glutathionylation of Fas.

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Inhibition of caspase-3 activity and activation by protein glutathionylation

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Glutathione depletion and disruption of intracellular ionic homeostasis regulate lymphoid cell apoptosis

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Mechanisms of reversible protein glutathionylation in redox signaling and oxidative stress

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Short interfering RNA-mediated silencing of glutaredoxin 2 increases the sensitivity of HeLa cells toward doxorubicin and phenylarsine oxide

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Overexpression of glutaredoxin 2 attenuates apoptosis by preventing cytochrome c release

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7 January 2021

Molecular mechanisms and potential clinical significance of S-glutathionylation.

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Protein glutathiolation by nitric oxide: an intracellular mechanism regulating redox protein modification

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Signalling by NO-induced protein S-nitrosylation and S-glutathionylation: convergences and divergences

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Protein S-nitrosylation: purview and parameters

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S-Nitrosylation of mitochondrial caspases

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Classification of cell death: recommendations of the Nomenclature Committee on Cell Death 2009

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Reactive oxygen species are essential for autophagy and specifically regulate the activity of Atg4

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LPS-induced autophagy is mediated by oxidative signaling in cardiomyocytes and is associated with cytoprotection

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Autophagy is Increased after Traumatic Brain Injury in Mice and is Partially Inhibited by the Antioxidant γ-glutamylcysteinyl Ethyl Ester

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The selection between apoptosis and necrosis is differentially regulated in hydrogen peroxide-treated and glutathione-depleted human promonocytic cells

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7 January 2021

Effect of ceramide on intracellular glutathione determines apoptotic or necrotic cell death of JB6 tumor cells

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7 January 2021

Glutathione in cancer biology and therapy

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BCL-2 and glutathione: alterations in cellular redox state that regulate apoptosis sensitivity

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Acceleration of glutathione efflux and inhibition of gamma-glutamyltranspeptidase sensitize metastatic B16 melanoma cells to endothelium-induced cytotoxicity.

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Multidrug resistance: retrospect and prospects in anti-cancer drug treatment.

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7 January 2021

Changes in thiol content and expression of glutathione redox system genes in the hippocampus and cerebellum in Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.107

7 January 2021

Role of glutathione in intracellular amyloid-alpha precursor protein/carboxy-terminal fragment aggregation and associated cytotoxicity

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.107

7 January 2021

Lack of glutathione peroxidase-1 exacerbates Abeta-mediated neurotoxicity in cortical neurons

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.107

7 January 2021

Overexpression of glutathione peroxidase increases the resistance of neuronal cells to Abeta-mediated neurotoxicity

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.107

7 January 2021

An increase in S-glutathionylated proteins in the Alzheimer's disease inferior parietal lobule, a proteomics approach.

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.107

7 January 2021

Involvement of glutaredoxin-1 and thioredoxin-1 in beta-amyloid toxicity and Alzheimer's disease.

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.107

7 January 2021

Indices of oxidative stress and mitochondrial function in individuals with incidental Lewy body disease

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.107

7 January 2021

Prevention of dopamine-induced cell death by thiol antioxidants: possible implications for treatment of Parkinson's disease.

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.107

7 January 2021

Lentivirus-mediated expression of glutathione peroxidase: neuroprotection in murine models of Parkinson's disease

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.107

7 January 2021

Regulation of mitochondrial NADP+-dependent isocitrate dehydrogenase activity by glutathionylation

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.107

7 January 2021

Glutaredoxin protects cerebellar granule neurons from dopamine-induced apoptosis by activating NF-kappa B via Ref-1.

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.107

7 January 2021

Intracellular signaling mechanisms of acetaminophen-induced liver cell death

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.107

7 January 2021

The glutathione precursor L-2-oxothiazolidine-4-carboxylic acid protects against liver injury due to chronic enteral ethanol exposure in the rat.

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.107

7 January 2021