Transcriptional upregulation of histone deacetylase 2 promotes Myc-induced oncogenic effects (Q38341800)

2 September 2017

Transcriptional upregulation of histone deacetylase 2 promotes Myc-induced oncogenic effects (English)

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2 September 2017

G M Marshall

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2 September 2017

S Gherardi

2

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2 September 2017

N Xu

3

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Z Neiron

4

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T Trahair

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C J Scarlett

6

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D K Chang

7

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P Y Liu

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K Jankowski

9

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N Iraci

10

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M Haber

11

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M D Norris

12

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J Keating

13

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E Sekyere

14

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G Jonquieres

15

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2 September 2017

F Stossi

16

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2 September 2017

B S Katzenellenbogen

17

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2 September 2017

A V Biankin

18

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G Perini

19

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T Liu

20

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2 September 2017

language of work or name

English

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publication date

9 August 2010

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2 September 2017

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29

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5957-5968

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44

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https://scigraph.springernature.com/pub.10.1038/onc.2010.332

2 September 2017

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Loss of acetylation at Lys16 and trimethylation at Lys20 of histone H4 is a common hallmark of human cancer

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HDAC2 mediates therapeutic resistance of pancreatic cancer cells via the BH3-only protein NOXA.

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Mechanisms of embryonal tumor initiation: distinct roles for MycN expression and MYCN amplification.

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Valproate inhibition of histone deacetylase 2 affects differentiation and decreases proliferation of endometrial stromal sarcoma cells

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Inhibition of histone deacetylase 2 increases apoptosis and p21Cip1/WAF1 expression, independent of histone deacetylase 1

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Myc-mediated transcriptional repression by recruitment of histone deacetylase.

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c-Myc represses transcription in vivo by a novel mechanism dependent on the initiator element and Myc box II

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Activation of tissue transglutaminase transcription by histone deacetylase inhibition as a therapeutic approach for Myc oncogenesis.

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Frequent amplification of 8q24, 11q, 17q, and 20q-specific genes in pancreatic cancer

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Two-step cross-linking method for identification of NF-kappaB gene network by chromatin immunoprecipitation.

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c-MYC: more than just a matter of life and death

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c-MYC activation in primary and metastatic ductal adenocarcinoma of the pancreas: incidence, mechanisms, and clinical significance

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Estrogen-occupied estrogen receptor represses cyclin G2 gene expression and recruits a repressor complex at the cyclin G2 promoter

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Class I histone deacetylase expression has independent prognostic impact in human colorectal cancer: specific role of class I histone deacetylases in vitro and in vivo

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Sp1 and Sp3 recruit histone deacetylase to repress transcription of human telomerase reverse transcriptase (hTERT) promoter in normal human somatic cells

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Cyclin G2 is degraded through the ubiquitin-proteasome pathway and mediates the antiproliferative effect of activin receptor-like kinase 7

31 March 2020

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Induction of HDAC2 expression upon loss of APC in colorectal tumorigenesis

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Reduced body size and decreased intestinal tumor rates in HDAC2-mutant mice.

31 March 2020

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31 March 2020

Identifiers

DOI

10.1038/ONC.2010.332

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Dimensions Publication ID

2 September 2017

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