The novel function of OCT4B isoform-265 in genotoxic stress (Q39411826)

11 September 2017

title

The novel function of OCT4B isoform-265 in genotoxic stress (English)

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11 September 2017

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Yuan Gao

1

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11 September 2017

Jianshu Wei

2

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11 September 2017

Jin Han

3

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11 September 2017

Xia Wang

4

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11 September 2017

Guannan Su

5

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11 September 2017

Yannan Zhao

6

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11 September 2017

Bing Chen

7

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11 September 2017

Zhifeng Xiao

8

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11 September 2017

Jiani Cao

9

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11 September 2017

Jianwu Dai

10

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11 September 2017

publication date

1 April 2012

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11 September 2017

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11 September 2017

volume

30

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11 September 2017

issue

4

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11 September 2017

page(s)

665-672

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Formation of pluripotent stem cells in the mammalian embryo depends on the POU transcription factor Oct4

11 September 2017

cites work

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Quantitative expression of Oct-3/4 defines differentiation, dedifferentiation or self-renewal of ES cells

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Core transcriptional regulatory circuitry in human embryonic stem cells

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Oct-4 knockdown induces similar patterns of endoderm and trophoblast differentiation markers in human and mouse embryonic stem cells

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Induced pluripotent stem cell lines derived from human somatic cells

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Induction of pluripotent stem cells from adult human fibroblasts by defined factors

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Reprogramming of human somatic cells to pluripotency with defined factors

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Human Oct3 gene family: cDNA sequences, alternative splicing, gene organization, chromosomal location, and expression at low levels in adult tissues

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OCT4 spliced variants are differentially expressed in human pluripotent and nonpluripotent cells.

21 January 2018

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Alternative translation of OCT4 by an internal ribosome entry site and its novel function in stress response.

21 January 2018

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The novel OCT4 spliced variant OCT4B1 can generate three protein isoforms by alternative splicing into OCT4B.

21 January 2018

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Oct-4 mRNA and protein expression during human preimplantation development

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POU5F1 isoforms show different expression patterns in human embryonic stem cells and preimplantation embryos

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The human OCT-4 isoforms differ in their ability to confer self-renewal

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Expression of transcription regulating genes in human preimplantation embryos

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Embryonic stem cell lines from human blastocysts: somatic differentiation in vitro

21 January 2018

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The transcriptome profile of human embryonic stem cells as defined by SAGE

21 January 2018

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Specific knockdown of Oct4 and beta2-microglobulin expression by RNA interference in human embryonic stem cells and embryonic carcinoma cells

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Molecular mechanisms of mammalian DNA repair and the DNA damage checkpoints

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p53-dependent signal transduction induced by stress

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Myc suppresses induction of the growth arrest genes gadd34, gadd45, and gadd153 by DNA-damaging agents

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Circadian proteins and genotoxic stress response

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Phosphorylation site interdependence of human p53 post-translational modifications in response to stress

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DNA damage, apoptosis and cell cycle changes induced by fluoride in rat oral mucosal cells and hepatocytes

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p53 in signaling checkpoint arrest or apoptosis

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Expression of transcription factor Oct4 in bladder cancer cell line T24 and its effects on the biological characteristics of the cells

21 January 2018

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Expression and significance of Oct4 in bladder cancer

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Oct4 expression in adult human stem cells: evidence in support of the stem cell theory of carcinogenesis

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The expression of pluripotency marker Oct 3/4 in prostate cancer and benign prostate hyperplasia

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Urinary thymidine dimer as a marker of total body burden of UV-inflicted DNA damage in humans

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Maintenance of genome stability in Saccharomyces cerevisiae

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The DNA damage response: putting checkpoints in perspective

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A mammalian cell cycle checkpoint pathway utilizing p53 and GADD45 is defective in ataxia-telangiectasia

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p53 controls both the G2/M and the G1 cell cycle checkpoints and mediates reversible growth arrest in human fibroblasts

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The spindle-assembly checkpoint: aiming for a perfect mitosis, every time

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BRCA1 physically associates with p53 and stimulates its transcriptional activity.

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Tumor suppressor p53 is a direct transcriptional activator of the human bax gene

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WAF1, a potential mediator of p53 tumor suppression

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21 January 2018

Identifiers

DOI

10.1002/STEM.1034

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11 September 2017

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