MicroRNA 483-3p suppresses the expression of DPC4/Smad4 in pancreatic cancer (Q39626698)

13 September 2017

MicroRNA 483-3p suppresses the expression of DPC4/Smad4 in pancreatic cancer (English)

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13 September 2017

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Jun Hao

1

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13 September 2017

Shuyu Zhang

2

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13 September 2017

Yingqi Zhou

3

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13 September 2017

Xiangui Hu

4

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13 September 2017

Chenghao Shao

5

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13 September 2017

language of work or name

English

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publication date

27 November 2010

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13 September 2017

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13 September 2017

585

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13 September 2017

207-213

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13 September 2017

1

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https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2010.11.039

13 September 2017

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Current diagnosis and treatment of pancreatic cancer in China

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Immunohistochemical labeling for dpc4 mirrors genetic status in pancreatic adenocarcinomas : a new marker of DPC4 inactivation

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Cooperative binding of Smad proteins to two adjacent DNA elements in the plasminogen activator inhibitor-1 promoter mediates transforming growth factor beta-induced smad-dependent transcriptional activation

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Direct binding of Smad3 and Smad4 to critical TGF beta-inducible elements in the promoter of human plasminogen activator inhibitor-type 1 gene

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Role of Smad proteins and transcription factor Sp1 in p21(Waf1/Cip1) regulation by transforming growth factor-beta.

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Smad4 and β-Catenin Co-activators Functionally Interact with Lymphoid-enhancing Factor to Regulate Graded Expression of Msx2

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A phylogenetically conserved cis-regulatory module in the Msx2 promoter is sufficient for BMP-dependent transcription in murine and Drosophila embryos

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Smad4 is dispensable for normal pancreas development yet critical in progression and tumor biology of pancreas cancer

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Smad4 is essential for down-regulation of E-cadherin induced by TGF-beta in pancreatic cancer cell line PANC-1

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CDKN2A, NF2, and JUN are dysregulated among other genes by miRNAs in malignant mesothelioma -A miRNA microarray analysis

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Oncogenic role of miR-483-3p at the IGF2/483 locus

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Identifiers

DOI

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13 September 2017

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