Cell division autoantigen 1 enhances signaling and the profibrotic effects of transforming growth factor-β in diabetic nephropathy (Q39641679)

13 September 2017

title

Cell division autoantigen 1 enhances signaling and the profibrotic effects of transforming growth factor-β in diabetic nephropathy (English)

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13 September 2017

Yugang Tu

1

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13 September 2017

Tieqiao Wu

2

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13 September 2017

Aozhi Dai

3

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13 September 2017

Yen Pham

4

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13 September 2017

Phyllis Chew

5

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13 September 2017

Judy B de Haan

6

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13 September 2017

Yu Wang

7

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13 September 2017

Ban-Hock Toh

8

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13 September 2017

Hongjian Zhu

9

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13 September 2017

Zemin Cao

10

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13 September 2017

Mark E Cooper

11

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13 September 2017

Zhonglin Chai

12

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13 September 2017

language of work or name

English

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publication date

20 October 2010

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13 September 2017

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13 September 2017

volume

79

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13 September 2017

issue

2

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13 September 2017

page(s)

199-209

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Extracellular matrix metabolism in diabetic nephropathy

13 September 2017

cites work

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Advanced glycation end products activate Smad signaling via TGF-beta-dependent and independent mechanisms: implications for diabetic renal and vascular disease

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Long-term prevention of renal insufficiency, excess matrix gene expression, and glomerular mesangial matrix expansion by treatment with monoclonal antitransforming growth factor-beta antibody in db/db diabetic mice

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Oral administration of GW788388, an inhibitor of TGF-beta type I and II receptor kinases, decreases renal fibrosis

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Connective tissue growth factor plays an important role in advanced glycation end product-induced tubular epithelial-to-mesenchymal transition: implications for diabetic renal disease.

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Cell division autoantigen 1 plays a profibrotic role by modulating downstream signalling of TGF-beta in a murine diabetic model of atherosclerosis

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7 January 2021

Disparate effects of angiotensin II antagonists and calcium channel blockers on albuminuria in experimental diabetes and hypertension: potential role of nephrin

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Renoprotective effects of vasopeptidase inhibition in an experimental model of diabetic nephropathy

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Cross-talk between ERK MAP kinase and Smad signaling pathways enhances TGF-beta-dependent responses in human mesangial cells

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7 January 2021

Connective tissue growth factor mediates transforming growth factor beta-induced collagen synthesis: down-regulation by cAMP

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7 January 2021

Connective tissue growth factor: potential role in glomerulosclerosis and tubulointerstitial fibrosis

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

Nephrin expression is reduced in human diabetic nephropathy: evidence for a distinct role for glycated albumin and angiotensin II.

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7 January 2021

Nephropathy in model combining genetic hypertension with experimental diabetes. Enalapril versus hydralazine and metoprolol therapy

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7 January 2021

A noninvasive computerized tail-cuff system for measuring blood pressure in mice

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7 January 2021

Accelerated nephropathy in diabetic apolipoprotein e-knockout mouse: role of advanced glycation end products.

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7 January 2021

Imatinib attenuates diabetic nephropathy in apolipoprotein E-knockout mice

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7 January 2021

Receptor for advanced glycation end products (RAGE) deficiency attenuates the development of atherosclerosis in diabetes

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7 January 2021

Protein kinase C beta inhibition attenuates the progression of experimental diabetic nephropathy in the presence of continued hypertension

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7 January 2021

Identifiers

DOI

10.1038/KI.2010.374

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13 September 2017

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