Synergistic up-regulation of Hexokinase-2, glucose transporters and angiogenic factors in pancreatic cancer cells by glucose deprivation and hypoxia. (Q40102972)

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English	Synergistic up-regulation of Hexokinase-2, glucose transporters and angiogenic factors in pancreatic cancer cells by glucose deprivation and hypoxia.	scientific article

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Synergistic up-regulation of Hexokinase-2, glucose transporters and angiogenic factors in pancreatic cancer cells by glucose deprivation and hypoxia (English)

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pancreatic cancer

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based on heuristic

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Mitsuteru Natsuizaka

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Mariko Ozasa

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Stephanie Darmanin

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Masaki Miyamoto

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Satoshi Kondo

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Suguru Kamada

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Masanobu Shindoh

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Fumihiro Higashino

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Wakako Suhara

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Hideyuki Koide

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Kiyoshi Aita

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Koji Nakagawa

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Takeshi Kondo

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Masahiro Asaka

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Masanobu Kobayashi

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29 June 2007

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Experimental Cell Research

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313

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3337-3348

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15

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2 January 2020

Hypoxia-inducible factor 1: oxygen homeostasis and disease pathophysiology

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7 January 2021

Perspectives on oxygen sensing

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The role of hypoxia inducible factor 1 (HIF-1) in hypoxia induced apoptosis

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Tumor hypoxia: definitions and current clinical, biologic, and molecular aspects

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Tumorigenesis and the angiogenic switch

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Role of angiogenesis in tumor growth and metastasis

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Angiogenesis as a therapeutic target

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The unfolding tale of the unfolded protein response

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The glucose-regulated proteins: stress induction and clinical applications

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Enhanced expression of asparagine synthetase under glucose-deprived conditions protects pancreatic cancer cells from apoptosis induced by glucose deprivation and cisplatin

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AMP-activated protein kinase signaling stimulates VEGF expression and angiogenesis in skeletal muscle

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5'-AMP-activated protein kinase (AMPK) is induced by low-oxygen and glucose deprivation conditions found in solid-tumor microenvironments

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AMP-activated protein kinase activity is critical for hypoxia-inducible factor-1 transcriptional activity and its target gene expression under hypoxic conditions in DU145 cells

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Glucose deprivation increases mRNA stability of vascular endothelial growth factor through activation of AMP-activated protein kinase in DU145 prostate carcinoma.

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The role of hypoxia-induced factors in tumor progression

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AMPK, the metabolic syndrome and cancer

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Complexes between the LKB1 tumor suppressor, STRAD alpha/beta and MO25 alpha/beta are upstream kinases in the AMP-activated protein kinase cascade

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TSC2 mediates cellular energy response to control cell growth and survival

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Cell cycle regulation via p53 phosphorylation by a 5'-AMP activated protein kinase activator, 5-aminoimidazole- 4-carboxamide-1-beta-D-ribofuranoside, in a human hepatocellular carcinoma cell line

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