JNK mediates TGF-beta1-induced epithelial mesenchymal transdifferentiation of mouse transformed keratinocytes (Q40229029)

18 September 2017

JNK mediates TGF-beta1-induced epithelial mesenchymal transdifferentiation of mouse transformed keratinocytes (English)

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18 September 2017

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Juan F Santibañez

1

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PubMed ID

16989819

18 September 2017

language of work or name

English

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publication date

12 September 2006

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18 September 2017

7

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based on heuristic

inferred from page(s)

published in

FEBS Letters

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18 September 2017

580

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18 September 2017

5385-5391

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18 September 2017

22

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18 September 2017

The Hallmarks of Cancer

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https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2006.09.003

Epithelial mesenchymal interactions in cancer and development

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Transcriptional control by the TGF-beta/Smad signaling system

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https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2006.09.003

Mechanisms of TGF-beta signaling from cell membrane to the nucleus

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https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2006.09.003

Smad-dependent and Smad-independent pathways in TGF-beta family signalling

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https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2006.09.003

Blockade of Smad4 in transformed keratinocytes containing a Ras oncogene leads to hyperactivation of the Ras-dependent Erk signalling pathway associated with progression to undifferentiated carcinomas

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1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2006.09.003

Involvement of the Ras/MAPK signaling pathway in the modulation of urokinase production and cellular invasiveness by transforming growth factor-beta(1) in transformed keratinocytes.

21 January 2018

1 reference

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Hematopoietic progenitor kinase 1 is a component of transforming growth factor beta-induced c-Jun N-terminal kinase signaling cascade

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1 reference

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The two faces of transforming growth factor beta in carcinogenesis

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TGFbeta signaling is necessary for carcinoma cell invasiveness and metastasis

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TGF-beta1 and Ha-Ras collaborate in modulating the phenotypic plasticity and invasiveness of epithelial tumor cells

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Transcriptional activation of the matrix metalloproteinase-9 gene in an H-ras and v-myc transformed rat embryo cell line

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The regulation of AP-1 activity by mitogen-activated protein kinases

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Metastasis is driven by sequential elevation of H-ras and Smad2 levels.

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Epithelial-mesenchymal transitions in tumour progression

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Induction and regulation of epithelial-mesenchymal transitions

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Requirement of TGF-beta receptor-dependent activation of c-Jun N-terminal kinases (JNKs)/stress-activated protein kinases (Sapks) for TGF-beta up-regulation of the urokinase-type plasminogen activator receptor.

21 January 2018

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Elucidation of Smad requirement in transforming growth factor-beta type I receptor-induced responses

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Transforming growth factor-beta1-dependent urokinase up-regulation and promotion of invasion are involved in Src-MAPK-dependent signaling in human ovarian cancer cells

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TGF-beta induces fibronectin synthesis through a c-Jun N-terminal kinase-dependent, Smad4-independent pathway

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The p150-Spir protein provides a link between c-Jun N-terminal kinase function and actin reorganization

21 January 2018

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Putting tumours in context

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The role of the cell-adhesion molecule E-cadherin as a tumour-suppressor gene

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Co-expression of tenascin-C and vimentin in human breast cancer cells indicates phenotypic transdifferentiation during tumour progression: correlation with histopathological parameters, hormone receptors, and oncoproteins

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1 reference

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10.1016/J.FEBSLET.2006.09.003

21 January 2018

Identifiers

DOI

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18 September 2017

1 reference

PubMed ID

16989819