Diallyl trisulfide-induced G(2)-M phase cell cycle arrest in human prostate cancer cells is caused by reactive oxygen species-dependent destruction and hyperphosphorylation of Cdc 25 C. (Q40412855)

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scientific article published on September 2005

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English	Diallyl trisulfide-induced G(2)-M phase cell cycle arrest in human prostate cancer cells is caused by reactive oxygen species-dependent destruction and hyperphosphorylation of Cdc 25 C.	scientific article published on September 2005

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

Diallyl trisulfide-induced G(2)-M phase cell cycle arrest in human prostate cancer cells is caused by reactive oxygen species-dependent destruction and hyperphosphorylation of Cdc 25 C. (English)

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

reactive oxygen species

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based on heuristic

inferred from title

prostate cancer

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based on heuristic

inferred from title

6

object named as

John S Lazo

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Europe PubMed Central

PubMed publication ID

20 September 2017

Anna Herman-Antosiewicz

2

object named as

Anna Herman-Antosiewicz

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Europe PubMed Central

PubMed publication ID

20 September 2017

Jedrzej Antosiewicz

3

object named as

Jedrzej Antosiewicz

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

Shivendra V Singh

7

object named as

Shivendra V Singh

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Europe PubMed Central

PubMed publication ID

20 September 2017

author name string

Dong Xiao

1

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

Hui Xiao

4

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Europe PubMed Central

PubMed publication ID

20 September 2017

Marni Brisson

5

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Europe PubMed Central

PubMed publication ID

20 September 2017

language of work or name

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publication date

1 September 2005

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Europe PubMed Central

PubMed publication ID

20 September 2017

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Europe PubMed Central

PubMed publication ID

20 September 2017

24

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Europe PubMed Central

PubMed publication ID

20 September 2017

41

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Europe PubMed Central

PubMed publication ID

20 September 2017

6256-6268

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Europe PubMed Central

PubMed publication ID

20 September 2017

Therapeutic actions of garlic constituents

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The Organosulfur Chemistry of the GenusAllium - Implications for the Organic Chemistry of Sulfur

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Suppression of tumorigenicity of human prostate carcinoma cells by replacing a mutated RB gene.

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Requirement for p53 and p21 to sustain G2 arrest after DNA damage

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p21-Mediated nuclear retention of cyclin B1-Cdk1 in response to genotoxic stress

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Down-regulation of prostasin serine protease: a potential invasion suppressor in prostate cancer

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Bax and Bak are required for apoptosis induction by sulforaphane, a cruciferous vegetable-derived cancer chemopreventive agent

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Diallyl disulfide (DADS) increases histone acetylation and p21(waf1/cip1) expression in human colon tumor cell lines

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Intermediate basal cells of the prostate: in vitro and in vivo characterization

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The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases

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Signal transduction pathways leading to cell cycle arrest and apoptosis induction in cancer cells by Allium vegetable-derived organosulfur compounds: a review

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Depressed p34cdc2 kinase activity and G2/M phase arrest induced by diallyl disulfide in HCT-15 cells

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Induction of apoptosis by diallyl disulfide through activation of caspase-3 in human leukemia HL-60 cells

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Isolation of diallyl trisulfide inducible differentially expressed genes in human gastric cancer cells by modified cDNA representational difference analysis

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Rapid destruction of human Cdc25A in response to DNA damage

1 reference

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Linkage of ATM to cell cycle regulation by the Chk2 protein kinase

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Phenotypic and genotypic characterization of commonly used human prostatic cell lines

1 reference

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Cell cycle checkpoints and their inactivation in human cancer

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Human Cdc25 A inactivation in response to S phase inhibition and its role in preventing premature mitosis

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Growth inhibitory effects of diallyl disulfide on human breast cancer cell lines

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Mitotic and G2 checkpoint control: regulation of 14-3-3 protein binding by phosphorylation of Cdc25C on serine-216

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Human cyclins A and B1 are differentially located in the cell and undergo cell cycle-dependent nuclear transport

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Effects of the garlic compound diallyl disulfide on the metabolism, adherence and cell cycle of HT-29 colon carcinoma cells: evidence of sensitive and resistant sub-populations

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Conservation of the Chk1 checkpoint pathway in mammals: linkage of DNA damage to Cdk regulation through Cdc25

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21 January 2018

Redox regulation of Cdc25C.

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Garlic and associated allyl sulfur components inhibit N-methyl-N-nitrosourea induced rat mammary carcinogenesis.

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Cyclin B1 expression in HeLa S3 cells studied by flow cytometry.

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Sulforaphane-induced G2/M phase cell cycle arrest involves checkpoint kinase 2-mediated phosphorylation of cell division cycle 25C.

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Effects of organosulfur compounds from garlic and onions on benzo[a]pyrene-induced neoplasia and glutathione S-transferase activity in the mouse.

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Diallyl disulfide induces apoptosis of human colon tumor cells

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Inhibitory effects of diallyl disulfide or aspirin on 2-amino-1-methyl-6-phenylimidazo[4,5-b]pyridine-induced mammary carcinogenesis in rats.

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10.1038/SJ.ONC.1208759

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

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PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

20 September 2017

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