Regulation of HtrA2/Omi by X-linked inhibitor of apoptosis protein in chemoresistance in human ovarian cancer cells (Q40428829)

20 September 2017

title

Regulation of HtrA2/Omi by X-linked inhibitor of apoptosis protein in chemoresistance in human ovarian cancer cells (English)

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20 September 2017

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6

Shixuan Wang

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20 September 2017

Xiaokui Yang

1

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20 September 2017

Hui Xing

2

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20 September 2017

Qinglei Gao

3

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20 September 2017

Gang Chen

4

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20 September 2017

Yunping Lu

5

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20 September 2017

Ding Ma

7

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20 September 2017

language of work or name

English

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publication date

1 May 2005

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20 September 2017

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20 September 2017

volume

97

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20 September 2017

issue

2

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20 September 2017

page(s)

413-421

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Prevention of apoptosis by a baculovirus gene during infection of insect cells

20 September 2017

cites work

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X-linked IAP is a direct inhibitor of cell-death proteases

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A novel anti-apoptosis gene, survivin, expressed in cancer and lymphoma

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Smac/DIABLO selectively reduces the levels of c-IAP1 and c-IAP2 but not that of XIAP and livin in HeLa cells

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Resistance of bone marrow-derived macrophages to apoptosis is associated with the expression of X-linked inhibitor of apoptosis protein in primary cultures of bone marrow cells

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Role of X-linked inhibitor of apoptosis protein in chemoresistance in ovarian cancer: possible involvement of the phosphoinositide-3 kinase/Akt pathway.

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A single BIR domain of XIAP sufficient for inhibiting caspases

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Antitumor agent parthenolide reverses resistance of breast cancer cells to tumor necrosis factor-related apoptosis-inducing ligand through sustained activation of c-Jun N-terminal kinase.

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Mitochondrial protease Omi/HtrA2 enhances caspase activation through multiple pathways

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Casein kinase I attenuates tumor necrosis factor-related apoptosis-inducing ligand-induced apoptosis by regulating the recruitment of fas-associated death domain and procaspase-8 to the death-inducing signaling complex

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The anti-apoptotic activity of XIAP is retained upon mutation of both the caspase 3- and caspase 9-interacting sites

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Reaper eliminates IAP proteins through stimulated IAP degradation and generalized translational inhibition

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Resistance to chemotherapy-induced apoptosis via decreased caspase-3 activity and overexpression of antiapoptotic proteins in ovarian cancer

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Doxorubicin induces apoptosis and CD95 gene expression in human primary endothelial cells through a p53-dependent mechanism.

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XIAP-mediated caspase inhibition in Hodgkin's lymphoma-derived B cells

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Interplay of Sp1 and Egr-1 in the proximal platelet-derived growth factor A-chain promoter in cultured vascular endothelial cells

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Resistance to apoptosis is correlated with the reduced caspase-3 activation and enhanced expression of antiapoptotic proteins in human cervical multidrug-resistant cells

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Cisplatin-induced CD95 redistribution into membrane lipid rafts of HT29 human colon cancer cells.

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Caspase inhibitors, but not c-Jun NH2-terminal kinase inhibitor treatment, prevent cisplatin-induced hearing loss.

7 January 2021

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Omi/HtrA2 protease mediates cisplatin-induced cell death in renal cells.

7 January 2021

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An apoptosis-inhibiting baculovirus gene with a zinc finger-like motif

7 January 2021

1 reference

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inferred from DOI database lookup

7 January 2021

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Identifiers

DOI

10.1016/J.YGYNO.2004.12.055

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20 September 2017

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