DSCR1 gene expression is dependent on NFATc1 during cardiac valve formation and colocalizes with anomalous organ development in trisomy 16 mice (Q40596613)

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scientific article published on February 2004

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English	DSCR1 gene expression is dependent on NFATc1 during cardiac valve formation and colocalizes with anomalous organ development in trisomy 16 mice	scientific article published on February 2004

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scholarly article

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Europe PubMed Central

21 September 2017

DSCR1 gene expression is dependent on NFATc1 during cardiac valve formation and colocalizes with anomalous organ development in trisomy 16 mice (English)

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Europe PubMed Central

21 September 2017

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inferred from title

Katherine E. Yutzey

object named as

Katherine E Yutzey

3

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Jeffery D. Molkentin

object named as

Jeffery D Molkentin

2

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author name string

Alexander W Lange

1

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Europe PubMed Central

21 September 2017

language of work or name

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publication date

1 February 2004

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Europe PubMed Central

21 September 2017

Developmental Biology

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Europe PubMed Central

21 September 2017

266

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Europe PubMed Central

21 September 2017

2

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Europe PubMed Central

21 September 2017

346-360

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Europe PubMed Central

21 September 2017

Nuclear localization of NF-ATc by a calcineurin-dependent, cyclosporin-sensitive intramolecular interaction

1 reference

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Abnormal blood vessel development and lethality in embryos lacking a single VEGF allele

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Dscr1, a novel endogenous inhibitor of calcineurin signaling, is expressed in the primitive ventricle of the heart and during neurogenesis.

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Cardiac compartment-specific overexpression of a modified retinoic acid receptor produces dilated cardiomyopathy and congestive heart failure in transgenic mice

1 reference

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NFAT signaling: choreographing the social lives of cells

1 reference

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Ocular findings in Down's syndrome

1 reference

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Role of the NF-ATc transcription factor in morphogenesis of cardiac valves and septum

1 reference

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VEGF modulates early heart valve formation.

1 reference

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Lack of regulation in the heart forming region of avian embryos.

1 reference

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Genetic control of the survival of murine trisomy 16 fetuses

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Mouse trisomy 16: an animal model of human trisomy 21 (Down syndrome)

1 reference

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Chronic overexpression of the calcineurin inhibitory gene DSCR1 (Adapt78) is associated with Alzheimer's disease

1 reference

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The DSCR1 (Adapt78) isoform 1 protein calcipressin 1 inhibits calcineurin and protects against acute calcium-mediated stress damage, including transient oxidative stress.

1 reference

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Heterozygous embryonic lethality induced by targeted inactivation of the VEGF gene

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A new human gene from the Down syndrome critical region encodes a proline-rich protein highly expressed in fetal brain and heart

1 reference

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Genomic organization, alternative splicing, and expression patterns of the DSCR1 (Down syndrome candidate region 1) gene

1 reference

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DSCR1, overexpressed in Down syndrome, is an inhibitor of calcineurin-mediated signaling pathways

1 reference

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Nf1 has an essential role in endothelial cells

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A gene expression map of human chromosome 21 orthologues in the mouse

1 reference

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NFAT signaling in vertebrate development

1 reference

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The DNA sequence of human chromosome 21

1 reference

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NFATc1 mediates vascular endothelial growth factor-induced proliferation of human pulmonary valve endothelial cells

1 reference

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Down syndrome and mouse models.

1 reference

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Down syndrome phenotypes: the consequences of chromosomal imbalance

1 reference

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Nkx-2.5 gene induction in mice is mediated by a Smad consensus regulatory region.

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Examination of the eyelid closure defect in trisomy 16 mice

1 reference

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The embryonic development of sensory organs and the skull in the trisomy 16 mouse, an animal model for Down's syndrome.

1 reference

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Partners in transcription: NFAT and AP-1.

1 reference

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7 January 2021

Trisomy 16 in the mouse fetus associated with generalized edema and cardiovascular and urinary tract anomalies.

1 reference

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Molecular cloning of a novel thyroid hormone-responsive gene, ZAKI-4, in human skin fibroblasts

1 reference

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A calcineurin-dependent transcriptional pathway for cardiac hypertrophy

1 reference

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A comparison of whole-genome shotgun-derived mouse chromosome 16 and the human genome.

1 reference

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Identification of amino acid residues and protein kinases involved in the regulation of NFATc subcellular localization

1 reference

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The transcription factor NF-ATc is essential for cardiac valve formation

1 reference

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Transcription factors of the NFAT family: regulation and function

1 reference

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Too much of a good thing: mechanisms of gene action in Down syndrome.

1 reference

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Human chromosome 21 gene expression atlas in the mouse.

1 reference

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A protein encoded within the Down syndrome critical region is enriched in striated muscles and inhibits calcineurin signaling

1 reference

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Myocyte-enriched calcineurin-interacting protein, MCIP1, inhibits cardiac hypertrophy in vivo

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The role of modulatory calcineurin-interacting proteins in calcineurin signaling

1 reference

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A new gene family including DSCR1 (Down Syndrome Candidate Region 1) and ZAKI-4: characterization from yeast to human and identification of DSCR1-like 2, a novel human member (DSCR1L2)

1 reference

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Dual roles of modulatory calcineurin-interacting protein 1 in cardiac hypertrophy

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Conotruncal anomalies in the trisomy 16 mouse: an immunohistochemical analysis with emphasis on the involvement of the neural crest

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Endocardial cushion development and heart loop architecture in the trisomy 16 mouse.

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Cardiac morphology at late fetal stages in the mouse with trisomy 16: consequences for different formation of the atrioventricular junction when compared to humans with trisomy 21

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Mechanisms of deficient cardiac septation in the mouse with trisomy 16.

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Occurrence of neuropathological changes and dementia of Alzheimer's disease in Down's syndrome.

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MEF2 responds to multiple calcium-regulated signals in the control of skeletal muscle fiber type

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Independent signals control expression of the calcineurin inhibitory proteins MCIP1 and MCIP2 in striated muscles

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Abnormal pinna type and hearing loss correlations in Down's syndrome.

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10.1016/J.YDBIO.2003.10.036

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Europe PubMed Central

21 September 2017

1 reference

Europe PubMed Central

21 September 2017

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