A highly conserved NTRK3 C-terminal sequence in the ETV6-NTRK3 oncoprotein binds the phosphotyrosine binding domain of insulin receptor substrate-1: an essential interaction for transformation (Q40608574)

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scientific article published on 9 December 2003

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English	A highly conserved NTRK3 C-terminal sequence in the ETV6-NTRK3 oncoprotein binds the phosphotyrosine binding domain of insulin receptor substrate-1: an essential interaction for transformation	scientific article published on 9 December 2003

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scholarly article

1 reference

Europe PubMed Central

22 September 2017

A highly conserved NTRK3 C-terminal sequence in the ETV6-NTRK3 oncoprotein binds the phosphotyrosine binding domain of insulin receptor substrate-1: an essential interaction for transformation (English)

1 reference

Europe PubMed Central

22 September 2017

1 reference

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author name string

Chris L Lannon

1

1 reference

Europe PubMed Central

22 September 2017

Matthew J Martin

2

1 reference

Europe PubMed Central

22 September 2017

Cristina E Tognon

3

1 reference

Europe PubMed Central

22 September 2017

Wook Jin

4

1 reference

Europe PubMed Central

22 September 2017

Seong-Jin Kim

5

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Europe PubMed Central

22 September 2017

Poul H B Sorensen

6

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Europe PubMed Central

22 September 2017

language of work or name

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publication date

9 December 2003

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Europe PubMed Central

22 September 2017

Journal of Biological Chemistry

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Europe PubMed Central

22 September 2017

279

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Europe PubMed Central

22 September 2017

8

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Europe PubMed Central

22 September 2017

6225-6234

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Europe PubMed Central

22 September 2017

Oncogenic kinase signalling

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A novel ETV6-NTRK3 gene fusion in congenital fibrosarcoma

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Congenital mesoblastic nephroma t(12;15) is associated with ETV6-NTRK3 gene fusion: cytogenetic and molecular relationship to congenital (infantile) fibrosarcoma

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Expression of the ETV6-NTRK3 gene fusion as a primary event in human secretory breast carcinoma

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The ETV6-NTRK3 gene fusion encodes a chimeric protein tyrosine kinase that transforms NIH3T3 cells

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Signal transduction and transforming properties of the TEL-TRKC fusions associated with t(12;15)(p13;q25) in congenital fibrosarcoma and acute myelogenous leukemia

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Naturally occurring tyrosine kinase inserts block high affinity binding of phospholipase C gamma and Shc to TrkC and neurotrophin-3 signaling

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ETV6-NTRK3 transformation requires insulin-like growth factor 1 receptor signaling and is associated with constitutive IRS-1 tyrosine phosphorylation

1 reference

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The insulin signalling system and the IRS proteins

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The pleckstrin homology domain is the principal link between the insulin receptor and IRS-1.

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PTB or not PTB -- that is the question

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Insulin signal transduction and the IRS proteins

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Role of insulin-like growth factors in embryonic and postnatal growth

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A TEL-JAK2 fusion protein with constitutive kinase activity in human leukemia

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Nucleophosmin-anaplastic lymphoma kinase of large-cell anaplastic lymphoma is a constitutively active tyrosine kinase that utilizes phospholipase C-gamma to mediate its mitogenicity

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Tissue-specific expression of the human tropomyosin gene involved in the generation of the trk oncogene

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Neurotrophin receptors, tumor progression and tumor maturation

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Neurotrophins and Trk receptors in human pancreatic ductal adenocarcinoma: expression patterns and effects on in vitro invasive behavior

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Identification and characterization of an activating TrkA deletion mutation in acute myeloid leukemia

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Rational basis for Trk inhibition therapy for prostate cancer.

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Gene expression of TrkC (NTRK3) in human soft tissue tumours

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Mutational analysis of the tyrosine kinome in colorectal cancers

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Insulin receptor substrate (IRS) transduction system: distinct and overlapping signaling potential

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Neoplastic transformation induced by insulin receptor substrate-1 overexpression requires an interaction with both Grb2 and Syp signaling molecules

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Overexpression of human insulin receptor substrate 1 induces cellular transformation with activation of mitogen-activated protein kinases

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IGF-I receptor signaling in a prostatic cancer cell line with a PTEN mutation

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BCR-ABL binds to IRS-1 and IRS-1 phosphorylation is inhibited by imatinib in K562 cells

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PTB domain binding to signaling proteins through a sequence motif containing phosphotyrosine

1 reference

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Distinct modes of interaction of SHC and insulin receptor substrate-1 with the insulin receptor NPEY region via non-SH2 domains

1 reference

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The phosphotyrosine interaction domain of SHC recognizes tyrosine-phosphorylated NPXY motif

1 reference

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PTEN inhibits insulin-stimulated MEK/MAPK activation and cell growth by blocking IRS-1 phosphorylation and IRS-1/Grb-2/Sos complex formation in a breast cancer model

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Insulin receptor substrate 1 translocation to the nucleus by the human JC virus T-antigen

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Nerve growth factor induced stimulation of Ras requires Trk interaction with Shc but does not involve phosphoinositide 3-OH kinase

1 reference

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Deletion of a conserved juxtamembrane sequence in Trk abolishes NGF-promoted neuritogenesis

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Role of neurotrophins and neurotrophins receptors in the in vitro invasion and heparanase production of human prostate cancer cells

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Identifiers

10.1074/JBC.M307388200

1 reference

Europe PubMed Central

22 September 2017

1 reference

Europe PubMed Central

22 September 2017

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