MycN sensitizes neuroblastoma cells for drug-induced apoptosis (Q40970050)

25 September 2017

MycN sensitizes neuroblastoma cells for drug-induced apoptosis (English)

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25 September 2017

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1

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25 September 2017

Klaus-Michael Debatin

series ordinal

4

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25 September 2017

Lutz W

2

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25 September 2017

Schwab M

series ordinal

3

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25 September 2017

language of work or name

English

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publication date

1 February 1999

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25 September 2017

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25 September 2017

18

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25 September 2017

1479-1486

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25 September 2017

7

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1202435

25 September 2017

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cites work

Overexpression of the death-promoting gene bax-alpha which is downregulated in breast cancer restores sensitivity to different apoptotic stimuli and reduces tumor growth in SCID mice

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Apoptotic cell death induced by c-myc is inhibited by bcl-2.

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Stress-induced Fas ligand expression in T cells is mediated through a MEK kinase 1-regulated response element in the Fas ligand promoter

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Apoptosis in cancer therapy: crossing the threshold

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Involvement of the CD95 (APO–1/Fas) receptor/ligand system in drug–induced apoptosis in leukemia cells

21 January 2018

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Mediation of c-Myc-induced apoptosis by p53.

21 January 2018

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Activation of CD95 (APO-1/Fas) signaling by ceramide mediates cancer therapy-induced apoptosis

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Requirement for the CD95 receptor-ligand pathway in c-Myc-induced apoptosis

21 January 2018

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Nuclear c-Myc plays an important role in the cytotoxicity of tumor necrosis factor alpha in tumor cells

21 January 2018

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Induction of TNF-sensitive cellular phenotype by c-Myc involves p53 and impaired NF-kappaB activation

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The proto-oncogene Bcl-2 and its role in regulating apoptosis

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Mitochondrial control of apoptosis

21 January 2018

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Requirement of an ICE/CED-3 protease for Fas/APO-1-mediated apoptosis

21 January 2018

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MycN and IFNgamma cooperate in apoptosis of human neuroblastoma cells.

21 January 2018

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FLICE is activated by association with the CD95 death-inducing signaling complex (DISC).

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A novel form of Epstein-Barr virus latency in normal B cells in vivo

21 January 2018

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FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

21 January 2018

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Drug-induced apoptosis in hepatoma cells is mediated by the CD95 (APO-1/Fas) receptor/ligand system and involves activation of wild-type p53.

21 January 2018

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Apoptosis by death factor

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A rapid and simple method for measuring thymocyte apoptosis by propidium iodide staining and flow cytometry

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Resistance to anticancer drugs in NIH3T3 cells transfected with c-myc and/or c-H-ras genes

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Two CD95 (APO-1/Fas) signaling pathways

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FLICE is predominantly expressed as two functionally active isoforms, caspase-8/a and caspase-8/b.

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The central executioner of apoptosis: multiple connections between protease activation and mitochondria in Fas/APO-1/CD95- and ceramide-induced apoptosis

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Monoclonal antibody-mediated tumor regression by induction of apoptosis.

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Movement of Bax from the cytosol to mitochondria during apoptosis

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FADD: essential for embryo development and signaling from some, but not all, inducers of apoptosis

21 January 2018

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Mitochondrial control of nuclear apoptosis.

21 January 2018

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c-Myc plays a role in cellular susceptibility to death receptor-mediated and chemotherapy-induced apoptosis in human monocytic leukemia U937 cells

21 January 2018

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7 January 2021

Chemosensitivity of solid tumor cellsin vitro is related to activation of the CD95 system

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Tumor suppressor p53 is a direct transcriptional activator of the human bax gene

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The overexpression of Bax produces cell death upon induction of the mitochondrial permeability transition

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7 January 2021

Identifiers

DOI

10.1038/SJ.ONC.1202435

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Dimensions Publication ID

25 September 2017

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