Mitogen-induced rapid phosphorylation of serine 795 of the retinoblastoma gene product in vascular smooth muscle cells involves ERK activation. (Q42459825)

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scientific article published on 6 April 2004

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English	Mitogen-induced rapid phosphorylation of serine 795 of the retinoblastoma gene product in vascular smooth muscle cells involves ERK activation.	scientific article published on 6 April 2004

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26 December 2019

Mitogen-induced rapid phosphorylation of serine 795 of the retinoblastoma gene product in vascular smooth muscle cells involves ERK activation (English)

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26 December 2019

phosphorylation

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based on heuristic

inferred from title

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based on heuristic

inferred from title

Maria Garnovskaya

1

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26 December 2019

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Yurii V Mukhin

2

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26 December 2019

Tamara M Vlasova

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26 December 2019

Jasjit S Grewal

4

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26 December 2019

Michael E Ullian

5

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26 December 2019

Baby G Tholanikunnel

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26 December 2019

John R Raymond

7

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26 December 2019

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6 April 2004

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26 December 2019

Journal of Biological Chemistry

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26 December 2019

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26 December 2019

23

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26 December 2019

24899-24905

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26 December 2019

Specificity of receptor tyrosine kinase signaling: transient versus sustained extracellular signal-regulated kinase activation

1 reference

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The retinoblastoma protein and cell cycle control

1 reference

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21 January 2018

Control of pRB phosphorylation

1 reference

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Disruption of retinoblastoma protein function by coexpression of its C pocket fragment.

1 reference

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Differential regulation of retinoblastoma protein function by specific Cdk phosphorylation sites

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Dual Mechanisms for the Inhibition of E2F Binding to RB by Cyclin-Dependent Kinase-Mediated RB Phosphorylation

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Control of the eukaryotic cell cycle by MAP kinase signaling pathways.

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p38 Mitogen-activated protein kinase is a critical component of the redox-sensitive signaling pathways activated by angiotensin II. Role in vascular smooth muscle cell hypertrophy

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The geranylgeranyltransferase I inhibitor GGTI-298 induces hypophosphorylation of retinoblastoma and partner switching of cyclin-dependent kinase inhibitors. A potential mechanism for GGTI-298 antitumor activity

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21 January 2018

Analysis of site-specific phosphorylation of the retinoblastoma protein during cell cycle progression.

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21 January 2018

p22phox is a critical component of the superoxide-generating NADH/NADPH oxidase system and regulates angiotensin II-induced hypertrophy in vascular smooth muscle cells

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Human retinoblastoma protein (Rb) is phosphorylated by cdc2 kinase and MAP kinase in Xenopus maturing oocytes.

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21 January 2018

Cyclin D1 expression is regulated positively by the p42/p44MAPK and negatively by the p38/HOGMAPK pathway.

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21 January 2018

Sustained activation of extracellular-signal-regulated kinase 1 (ERK1) is required for the continued expression of cyclin D1 in G1 phase

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Assembly of cyclin D-dependent kinase and titration of p27Kip1 regulated by mitogen-activated protein kinase kinase (MEK1)

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21 January 2018

Inhibition of ras-induced proliferation and cellular transformation by p16INK4.

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21 January 2018

Angiotensin II activation of cyclin D1-dependent kinase activity

1 reference

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21 January 2018

Ras signalling linked to the cell-cycle machinery by the retinoblastoma protein

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21 January 2018

Myc and Ras collaborate in inducing accumulation of active cyclin E/Cdk2 and E2F.

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Rac is required for v-Abl tyrosine kinase to activate mitogenesis

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21 January 2018

Cooperating oncogenes converge to regulate cyclin/cdk complexes.

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21 January 2018

Convergence of mitogenic signalling cascades from diverse classes of receptors at the cyclin D-cyclin-dependent kinase-pRb-controlled G1 checkpoint

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21 January 2018

Activation of p70 S6 Kinase and erk-encoded Mitogen-activated Protein Kinases Is Resistant to High Cyclic Nucleotide Levels in Swiss 3T3 Fibroblasts

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Rapamycin-FKBP inhibits cell cycle regulators of proliferation in vascular smooth muscle cells

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Activation of phosphatidylinositol 3-kinase is sufficient for cell cycle entry and promotes cellular changes characteristic of oncogenic transformation

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Defining the minimal portion of the retinoblastoma protein that serves as an efficient substrate for cdk4 kinase/cyclin D1 complex

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Heterodimerization of the transcription factors E2F-1 and DP-1 leads to cooperative trans-activation

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