Arsenic-induced promoter hypomethylation and over-expression of ERCC2 reduces DNA repair capacity in humans by non-disjunction of the ERCC2-Cdk7 complex. (Q42707122)

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scientific article published on April 2014

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English	Arsenic-induced promoter hypomethylation and over-expression of ERCC2 reduces DNA repair capacity in humans by non-disjunction of the ERCC2-Cdk7 complex.	scientific article published on April 2014

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scholarly article

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6 March 2020

Arsenic-induced promoter hypomethylation and over-expression of ERCC2 reduces DNA repair capacity in humans by non-disjunction of the ERCC2-Cdk7 complex (English)

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6 March 2020

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author name string

Nilanjana Banerjee

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Aditi Chatterjee

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Tanmoy J Sau

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Jayanta K Das

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Prafulla K Mishra

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Partha Chakrabarti

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Arun Bandyopadhyay

8

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Ashok K Giri

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publication date

1 April 2014

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4

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864-873

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6 March 2020

Arsenic-induced toxicity and carcinogenicity: a two-wave cross-sectional study in arsenicosis individuals in West Bengal, India.

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Liver is a target of arsenic carcinogenesis

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Chronic inorganic arsenic exposure induces hepatic global and individual gene hypomethylation: implications for arsenic hepatocarcinogenesis

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DNA hypermethylation of promoter of gene p53 and p16 in arsenic-exposed people with and without malignancy

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Polymorphism in the ERCC2 codon 751 is associated with arsenic-induced premalignant hyperkeratosis and significant chromosome aberrations.

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Mutations in XPB and XPD helicases found in xeroderma pigmentosum patients impair the transcription function of TFIIH.

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The CDK7-cycH-p36 complex of transcription factor IIH phosphorylates p53, enhancing its sequence-specific DNA binding activity in vitro

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XPB and XPD helicases in TFIIH orchestrate DNA duplex opening and damage verification to coordinate repair with transcription and cell cycle via CAK kinase.

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A yeast four-hybrid system identifies Cdk-activating kinase as a regulator of the XPD helicase, a subunit of transcription factor IIH

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Nucleotide excision repair driven by the dissociation of CAK from TFIIH.

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On the traces of XPD: cell cycle matters - untangling the genotype-phenotype relationship of XPD mutations

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Tissue-specific accelerated aging in nucleotide excision repair deficiency

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Xpd/Ercc2 regulates CAK activity and mitotic progression

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Arsenic exposure and DNA methylation among elderly men.

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Interdependent genotoxic mechanisms of monomethylarsonous acid: role of ROS-induced DNA damage and poly(ADP-ribose) polymerase-1 inhibition in the malignant transformation of urothelial cells

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Both hypomethylation and hypermethylation of DNA associated with arsenite exposure in cultures of human cells identified by methylation-sensitive arbitrarily-primed PCR.

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Evaluation of micronucleus induction in a Chilean population environmentally exposed to arsenic.

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A novel cyclin associates with MO15/CDK7 to form the CDK-activating kinase

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p53 is phosphorylated by CDK7-cyclin H in a p36MAT1-dependent manner

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Identifiers

10.1039/C3MT00328K

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Europe PubMed Central

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6 March 2020

1 reference

Europe PubMed Central

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6 March 2020

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