Expression of Rac1b stimulates NF-kappaB-mediated cell survival and G1/S progression (Q42816126)

7 November 2017

Expression of Rac1b stimulates NF-kappaB-mediated cell survival and G1/S progression (English)

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7 November 2017

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7 November 2017

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PubMed ID

15817154

7 November 2017

language of work or name

English

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publication date

1 May 2005

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Experimental Cell Research

7 November 2017

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7 November 2017

305

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7 November 2017

2

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7 November 2017

292-299

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Rho GTPases and the actin cytoskeleton

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The small GTP-binding proteins Rac1 and Cdc42 regulate the activity of the JNK/SAPK signaling pathway

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Selective activation of the JNK signaling cascade and c-Jun transcriptional activity by the small GTPases Rac and Cdc42Hs

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Activation of the nuclear factor-kappaB by Rho, CDC42, and Rac-1 proteins

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Integration of Rac-dependent regulation of cyclin D1 transcription through a nuclear factor-kappaB-dependent pathway

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NF-kappaB in cancer: from innocent bystander to major culprit

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NF-kappaB function in growth control: regulation of cyclin D1 expression and G0/G1-to-S-phase transition

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NF-kappaB controls cell growth and differentiation through transcriptional regulation of cyclin D1.

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Suppression of Ras-induced apoptosis by the Rac GTPase

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Cloning of a novel human Rac1b splice variant with increased expression in colorectal tumors

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Rac1 in human breast cancer: overexpression, mutation analysis, and characterization of a new isoform, Rac1b

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Small GTPase Rac1: structure, localization, and expression of the human gene.

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Alternative splicing of Rac1 generates Rac1b, a self-activating GTPase

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Calcium signaling regulates translocation and activation of Rac.

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An essential role for Rho, Rac, and Cdc42 GTPases in cell cycle progression through G1

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Rac1 is required for cell proliferation and G2/M progression.

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TEL, a putative tumor suppressor, induces apoptosis and represses transcription of Bcl-XL.

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PKCη associates with cyclin E/Cdk2 complex in serum-starved MCF-7 and NIH-3T3 cells

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A phosphatidylinositol 3-kinase docking site in the cytoplasmic tail of the Jaagsiekte sheep retrovirus transmembrane protein is essential for envelope-induced transformation of NIH 3T3 cells

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The v-Crk oncogene enhances cell survival and induces activation of protein kinase B/Akt.

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Comparison of proliferating cell nuclear antigen (PCNA) staining and BrdUrd-labelling index under different proliferative conditions in vitro by flow cytometry

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To cycle or not to cycle: a critical decision in cancer.

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Rac GTPase instructs nuclear factor-kappaB activation by conveying the SCF complex and IkBalpha to the ruffling membranes

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An essential role for Rac in Ras transformation

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Matrix-dependent Tiam1/Rac signaling in epithelial cells promotes either cell-cell adhesion or cell migration and is regulated by phosphatidylinositol 3-kinase

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Phosphotyrosine-dependent activation of Rac-1 GDP/GTP exchange by the vav proto-oncogene product

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Mice deficient in the Rac activator Tiam1 are resistant to Ras-induced skin tumours.

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Interaction of Rac with p67phox and regulation of phagocytic NADPH oxidase activity

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Rac "insert region" is a novel effector region that is implicated in the activation of NADPH oxidase, but not PAK65

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Mutational analysis of novel effector domains in Rac1 involved in the activation of nicotinamide adenine dinucleotide phosphate (reduced) oxidase

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Structure of the TPR domain of p67phox in complex with Rac.GTP

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10.1016/J.YEXCR.2004.12.029

7 January 2021

Identifiers

DOI

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7 November 2017

1 reference

PubMed ID

15817154