Regulation of amyloid precursor protein (APP) phosphorylation and processing by p35/Cdk5 and p25/Cdk5. (Q44157383)

30 November 2017

title

Regulation of amyloid precursor protein (APP) phosphorylation and processing by p35/Cdk5 and p25/Cdk5. (English)

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30 November 2017

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Feng Liu

1

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30 November 2017

Yuan Su

2

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30 November 2017

Baolin Li

3

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30 November 2017

Yan Zhou

4

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30 November 2017

John Ryder

5

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30 November 2017

Patricia Gonzalez-DeWhitt

6

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30 November 2017

Patrick C May

7

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30 November 2017

Binhui Ni

8

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30 November 2017

language of work or name

English

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publication date

1 July 2003

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30 November 2017

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30 November 2017

volume

547

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30 November 2017

issue

1-3

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30 November 2017

page(s)

193-196

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Alzheimer's disease: Initial report of the purification and characterization of a novel cerebrovascular amyloid protein

30 November 2017

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Conversion of p35 to p25 deregulates Cdk5 activity and promotes neurodegeneration

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Mutation of the beta-amyloid precursor protein in familial Alzheimer's disease increases beta-protein production

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Effect of protein kinase A inhibitors on the production of Abeta40 and Abeta42 by human cells expressing normal and Alzheimer's disease-linked mutated betaAPP and presenilin 1.

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The cytoplasmic domain of Alzheimer's amyloid precursor protein is phosphorylated at Thr654, Ser655, and Thr668 in adult rat brain and cultured cells.

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Role of phosphorylation of Alzheimer's amyloid precursor protein during neuronal differentiation.

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Cyclin-dependent kinase 5-deficient mice demonstrate novel developmental arrest in cerebral cortex

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10.1016/S0014-5793(03)00714-2

7 January 2021

Identifiers

DOI

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30 November 2017

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