Inhibition of interferon-gamma-activated nuclear factor-kappa B by cyclosporin A: A possible mechanism for synergistic induction of apoptosis by interferon-gamma and cyclosporin A in gastric carcinoma cells (Q44454781)

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scientific article published in June 2003

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English	Inhibition of interferon-gamma-activated nuclear factor-kappa B by cyclosporin A: A possible mechanism for synergistic induction of apoptosis by interferon-gamma and cyclosporin A in gastric carcinoma cells	scientific article published in June 2003

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scholarly article

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Europe PubMed Central

PubMed publication ID

3 December 2017

Inhibition of interferon-gamma-activated nuclear factor-kappa B by cyclosporin A: A possible mechanism for synergistic induction of apoptosis by interferon-gamma and cyclosporin A in gastric carcinoma cells (English)

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Europe PubMed Central

PubMed publication ID

3 December 2017

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based on heuristic

inferred from title

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author name string

Kiichiro Beppu

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3 December 2017

Takashi Morisaki

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3 December 2017

Hisashi Matsunaga

3

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3 December 2017

Akihiko Uchiyama

4

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3 December 2017

Hideo Kanaide

7

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Europe PubMed Central

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3 December 2017

Masao Tanaka

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3 December 2017

Mitsuo Katano

9

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3 December 2017

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publication date

1 June 2003

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3 December 2017

Biochemical and Biophysical Research Communications

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3 December 2017

305

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4

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3 December 2017

797-805

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3 December 2017

Activation of the STAT signaling pathway can cause expression of caspase 1 and apoptosis

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Jak-STAT pathways and transcriptional activation in response to IFNs and other extracellular signaling proteins

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Contribution of STAT SH2 groups to specific interferon signaling by the Jak-STAT pathway

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Loss of STAT1 expression confers resistance to IFN-gamma-induced apoptosis in ME180 cells

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Formation of STAT1-STAT2 heterodimers and their role in the activation of IRF-1 gene transcription by interferon-alpha

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Calcium signaling

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Control of apoptosis by Rel/NF-kappaB transcription factors

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Calcineurin is a common target of cyclophilin-cyclosporin A and FKBP-FK506 complexes

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The mechanism of action of cyclosporin A and FK506

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Calcineurin acts in synergy with PMA to inactivate I kappa B/MAD3, an inhibitor of NF-kappa B.

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7 January 2021

Cyclosporine A is an uncompetitive inhibitor of proteasome activity and prevents NF-kappaB activation

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Enhancement by captopril of bradykinin-induced calcium transients in cultured endothelial cells of the bovine aorta

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Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

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Lipopolysaccharide increases cyclo-oxygenase-2 expression in a colon carcinoma cell line through nuclear factor-kappa B activation

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In vivo transfection of cis element "decoy" against nuclear factor-kappaB binding site prevents myocardial infarction

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Evidence that de novo protein synthesis is dispensable for anti-apoptotic effects of NF-kappaB.

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7 January 2021

Differential activation of transcription factors induced by Ca2+ response amplitude and duration

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Defective TNF-alpha-induced apoptosis in STAT1-null cells due to low constitutive levels of caspases

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7 January 2021

Targeted disruption of the Stat1 gene in mice reveals unexpected physiologic specificity in the JAK-STAT signaling pathway

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7 January 2021

Unresponsiveness to interferon associated with STAT1 protein deficiency in a gastric adenocarcinoma cell line.

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IFNgamma sensitizes for apoptosis by upregulating caspase-8 expression through the Stat1 pathway.

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RNA-dependent protein kinase PKR is required for activation of NF-kappa B by IFN-gamma in a STAT1-independent pathway.

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The Jak-STAT pathway

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Critical role of tumor necrosis factor-alpha and NF-kappa B in interferon-gamma -induced CD40 expression in microglia/macrophages

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NF-kappa B activation mediates the cross-talk between extracellular matrix and interferon-gamma (IFN-gamma) leading to enhanced monokine induced by IFN-gamma (MIG) expression in macrophages

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Identifiers

10.1016/S0006-291X(03)00853-2

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Europe PubMed Central

PubMed publication ID

3 December 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

3 December 2017

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