Mechanism for quinolinic acid cytotoxicity in human astrocytes and neurons. (Q45969395)

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31 January 2020

title

Mechanism for quinolinic acid cytotoxicity in human astrocytes and neurons (English)

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31 January 2020

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Nady Braidy

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Seray Adams

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Bruce J Brew

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31 January 2020

author name string

Ross Grant

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31 January 2020

publication date

18 April 2009

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31 January 2020

published in

Neurotoxicity Research

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31 January 2020

volume

16

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31 January 2020

page(s)

77-86

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31 January 2020

issue

1

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https://scigraph.springernature.com/pub.10.1007/s12640-009-9051-z

31 January 2020

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cites work

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Endogenous neurotoxins from tryptophan

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Poly(ADP-ribose) polymerase-1 is involved in the neuronal death induced by quinolinic acid in rats

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Poly(ADP-ribose) polymerase is a mediator of necrotic cell death by ATP depletion

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In vitro ischemia promotes glutamate-mediated free radical generation and intracellular calcium accumulation in hippocampal pyramidal neurons.

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Blockade of quinolinic acid-induced neurotoxicity by pyruvate is associated with inhibition of glial activation in a model of Huntington's disease

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Apoptotic neuron death in rat substantia nigra induced by striatal excitotoxic injury is developmentally dependent.

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Oxidative stress as a mechanism for quinolinic acid-induced hippocampal damage: protection by melatonin and deprenyl

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The anticonvulsant MK-801 is a potent N-methyl-D-aspartate antagonist

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Murine glial cells regenerate NAD, after peroxide-induced depletion, using either nicotinic acid, nicotinamide, or quinolinic acid as substrates

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Direct Quantitation of Poly(ADP-Ribose) Polymerase (PARP) Activity as a Means to Distinguish Necrotic and Apoptotic Death in Cell and Tissue Samples

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In vivo hydroxyl radical formation after quinolinic acid infusion into rat corpus striatum

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Expression of indoleamine 2,3-dioxygenase and production of quinolinic acid by human microglia, astrocytes, and neurons

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The excitotoxin quinolinic acid is present in the brain of several mammals and its cortical content increases during the aging process

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Quinolinic acid selectively induces apoptosis of human astrocytes: potential role in AIDS dementia complex.

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Nitric oxide activation of poly(ADP-ribose) synthetase in neurotoxicity

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10.1007/S12640-009-9051-Z

Identifiers

DOI

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19526301%20AND%20SRC:MED&resulttype=core&format=json

Dimensions Publication ID

31 January 2020

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