Efficient execution of cell death in non-glycolytic cells requires the generation of ROS controlled by the activity of mitochondrial H+-ATP synthase (Q46856395)

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scientific article published on 16 December 2005

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English	Efficient execution of cell death in non-glycolytic cells requires the generation of ROS controlled by the activity of mitochondrial H+-ATP synthase	scientific article published on 16 December 2005

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scholarly article

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Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

Efficient execution of cell death in non-glycolytic cells requires the generation of ROS controlled by the activity of mitochondrial H+-ATP synthase (English)

1 reference

Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

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Isabel Fabregat

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Isabel Fabregat

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Gema Santamaría

1

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Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

Marta Martínez-Diez

2

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Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

José M Cuezva

4

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Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

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16 December 2005

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Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

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Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

27

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Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

925-935

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Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

5

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Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

Mitochondrial diseases in man and mouse

1 reference

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Organelle-specific initiation of cell death pathways

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BAD and glucokinase reside in a mitochondrial complex that integrates glycolysis and apoptosis

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In self-defence: hexokinase promotes voltage-dependent anion channel closure and prevents mitochondria-mediated apoptotic cell death

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AIF deficiency compromises oxidative phosphorylation

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Bcl-2 blocks apoptosis in cells lacking mitochondrial DNA.

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Activation of caspases occurs downstream from radical oxygen species production, Bcl-xL down-regulation, and early cytochrome C release in apoptosis induced by transforming growth factor beta in rat fetal hepatocytes

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21 January 2018

Reactive oxygen species (ROS) mediates the mitochondrial-dependent apoptosis induced by transforming growth factor (beta) in fetal hepatocytes.

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Changes in intramitochondrial and cytosolic pH: early events that modulate caspase activation during apoptosis

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Bafilomycins: a class of inhibitors of membrane ATPases from microorganisms, animal cells, and plant cells

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Triggering caspase-independent cell death to combat cancer.

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Mitochondria in cell death: novel targets for neuroprotection and cardioprotection

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Stress-induced activation of the p53 tumor suppressor in leukemia cells and normal lymphocytes requires mitochondrial activity and reactive oxygen species.

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Preservation of mitochondrial structure and function after Bid- or Bax-mediated cytochrome c release

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The role of dynamin-related protein 1, a mediator of mitochondrial fission, in apoptosis

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DRP-1-mediated mitochondrial fragmentation during EGL-1-induced cell death in C. elegans

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Identifiers

10.1093/CARCIN/BGI315

1 reference

Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

1 reference

Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

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