ERK phosphorylation and miR-181a expression modulate activation of human memory TH17 cells. (Q50746618)

18 March 2018

title

ERK phosphorylation and miR-181a expression modulate activation of human memory TH17 cells. (English)

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18 March 2018

1 reference

Dominik Aschenbrenner

4

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18 March 2018

Silvia Monticelli

9

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18 March 2018

Simone Becattini

Simone Becattini

5

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Daniela Latorre

Daniela Latorre

6

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Federica Sallusto

8

Federica Sallusto

1 reference

18 March 2018

7

Antonio Lanzavecchia

1 reference

18 March 2018

2

Camilla Basso

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18 March 2018

Federico Mele

1

Federico Mele

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18 March 2018

author name string

Cristina Leoni

3

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18 March 2018

language of work or name

English

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publication date

16 March 2015

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18 March 2018

published in

Nature Communications

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18 March 2018

volume

6

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18 March 2018

page(s)

6431

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18 March 2018

https://scigraph.springernature.com/pub.10.1038/ncomms7431

exact match

0 references

1 reference

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Recent advances in understanding the molecular mechanisms of the development and function of Th17 cells

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The effect of antigen dose on CD4+ T helper cell phenotype development in a T cell receptor-alpha beta-transgenic model

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Low-strength T-cell activation promotes Th17 responses.

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The role of miRNAs in mast cells and other innate immune cells

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MicroRNAs in T helper cell differentiation and plasticity

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An emerging player in the adaptive immune response: microRNA-146a is a modulator of IL-2 expression and activation-induced cell death in T lymphocytes.

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miR-181a is an intrinsic modulator of T cell sensitivity and selection

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MiR-146a and NF-κB1 regulate mast cell survival and T lymphocyte differentiation

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miR-146a controls the resolution of T cell responses in mice

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Dynamic regulation of miRNA expression in ordered stages of cellular development.

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An endogenous positively selecting peptide enhances mature T cell responses and becomes an autoantigen in the absence of microRNA miR-181a

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Surface phenotype and antigenic specificity of human interleukin 17-producing T helper memory cells

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Interleukins 1beta and 6 but not transforming growth factor-beta are essential for the differentiation of interleukin 17-producing human T helper cells

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Pathogen-induced human TH17 cells produce IFN-γ or IL-10 and are regulated by IL-1β.

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Decline in miR-181a expression with age impairs T cell receptor sensitivity by increasing DUSP6 activity

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Human neonatal naive CD4+ T cells have enhanced activation-dependent signaling regulated by the microRNA miR-181a

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Dusp5 negatively regulates IL-33-mediated eosinophil survival and function

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Differential regulation of MAP kinase signalling by dual-specificity protein phosphatases.

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Memory and flexibility of cytokine gene expression as separable properties of human T(H)1 and T(H)2 lymphocytes

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Opposing regulation of the locus encoding IL-17 through direct, reciprocal actions of STAT3 and STAT5

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Interleukin-2 signaling via STAT5 constrains T helper 17 cell generation

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Interplay between RORgammat, Egr3, and E proteins controls proliferation in response to pre-TCR signals.

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Impaired immune responses and B-cell proliferation in mice lacking the Id3 gene

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Control of the differentiation of regulatory T cells and T(H)17 cells by the DNA-binding inhibitor Id3.

21 January 2018

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Regulation of the helix-loop-helix proteins, E2A and Id3, by the Ras-ERK MAPK cascade

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The dual nature of TH17 cells: shifting the focus to function

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Dwelling on T cell fate decisions

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MicroRNA miR-326 regulates TH-17 differentiation and is associated with the pathogenesis of multiple sclerosis

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MicroRNA-155 as a proinflammatory regulator in clinical and experimental arthritis

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MicroRNA-301a regulation of a T-helper 17 immune response controls autoimmune demyelination

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MicroRNA-155 promotes autoimmune inflammation by enhancing inflammatory T cell development

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Next-generation sequencing identifies microRNAs that associate with pathogenic autoimmune neuroinflammation in rats

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Phosphatase-mediated crosstalk between MAPK signaling pathways in the regulation of cell survival

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Regulation of TH17 cell differentiation by innate immune signals

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ERK-dependent T cell receptor threshold calibration in rheumatoid arthritis

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NFAT1 and NFAT2 are positive regulators of IL-4 gene transcription

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Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

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Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

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The interplay between the duration of TCR and cytokine signaling determines T cell polarization

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inferred from DOI database lookup

7 January 2021

based on heuristic

Hepatitis C virus-induced reduction in miR-181a impairs CD4(+) T-cell responses through overexpression of DUSP6.

1 reference

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inferred from DOI database lookup

7 January 2021

based on heuristic

Induced recruitment of NK cells to lymph nodes provides IFN-gamma for T(H)1 priming

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inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1038/NCOMMS7431

1 reference

18 March 2018

0 references

1 reference

18 March 2018