Acute lymphoblastic leukemia with t(4;11) in children 1 year and older: The 'big sister' of the infant disease? (Q50775080)

19 March 2018

title

Acute lymphoblastic leukemia with t(4;11) in children 1 year and older: The 'big sister' of the infant disease? (English)

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19 March 2018

main subject

lymphoblastic leukemia

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inferred from title

author

Alexander Michael Dohnal

8

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author name string

Mann G

1

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Cazzaniga G

2

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van der Velden VH

3

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Flohr T

4

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Csinady E

5

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Paganin M

6

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Schrauder A

7

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Schrappe M

9

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Biondi A

10

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Gadner H

11

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van Dongen JJ

12

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Panzer-Grümayer ER

13

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publication date

8 February 2007

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19 March 2018

published in

Leukemia

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volume

21

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issue

4

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19 March 2018

page(s)

642-646

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Molecular mechanisms of leukemogenesis mediated by MLL fusion proteins

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Clinical heterogeneity in childhood acute lymphoblastic leukemia with 11q23 rearrangements

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Biased distribution of chromosomal breakpoints involving the MLL gene in infants versus children and adults with t(4;11) ALL.

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7 January 2021

Molecular monitoring of residual disease using antigen receptor genes in childhood acute lymphoblastic leukaemia.

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Ig gene rearrangement steps are initiated in early human precursor B cell subsets and correlate with specific transcription factor expression

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Immunoglobulin light chain gene rearrangements display hierarchy in absence of selection for functionality in precursor-B-ALL

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High incidence and unique features of antigen receptor gene rearrangements in TEL-AML1-positive leukemias.

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Standardization and quality control studies of 'real-time' quantitative reverse transcriptase polymerase chain reaction of fusion gene transcripts for residual disease detection in leukemia - a Europe Against Cancer program.

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Split-signal FISH for detection of chromosome aberrations in acute lymphoblastic leukemia

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B precursor acute lymphoblastic leukemia third complementarity-determining regions predominantly represent an unbiased recombination repertoire: leukemic transformation frequently occurs in fetal life

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Site-specific translocation and evidence of postnatal origin of the t(1;19) E2A-PBX1 fusion in childhood acute lymphoblastic leukemia

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Rearrangement status of the malignant cell determines type of secondary IgH rearrangement (V-replacement or V to DJ joining) in childhood B precursor acute lymphoblastic leukemia

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7 January 2021

MLL rearrangements are induced by low doses of etoposide in human fetal hematopoietic stem cells

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7 January 2021

MLL chimeric protein activation renders cells vulnerable to chromosomal damage: an explanation for the very short latency of infant leukemia

1 reference

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7 January 2021

Presence of N regions in the clonotypic DJ rearrangements of the immunoglobulin heavy-chain genes indicates an exquisitely short latency in t(4;11)-positive infant acute lymphoblastic leukemia.

1 reference

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7 January 2021

Pro-B-cell to pre-B-cell development in B-lineage acute lymphoblastic leukemia expressing the MLL/AF4 fusion protein

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7 January 2021

Identifiers

DOI

10.1038/SJ.LEU.2404577

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19 March 2018

Dimensions Publication ID

1002878947

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19 March 2018