VapB as a regulator of osteoclastogenesis via modulation of PLCγ2-Ca(2+)-NFAT signaling. (Q53391310)

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20 January 2020

title

VapB as a regulator of osteoclastogenesis via modulation of PLCγ2-Ca(2+)-NFAT signaling (English)

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20 January 2020

1

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20 January 2020

author name string

Jeong-Tae Yeon

2

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20 January 2020

Kie-In Park

3

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20 January 2020

Chang Hoon Lee

4

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20 January 2020

Byung Soo Youn

5

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20 January 2020

Jaemin Oh

6

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20 January 2020

Myeung Su Lee

7

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20 January 2020

language of work or name

English

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publication date

10 January 2012

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20 January 2020

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20 January 2020

volume

586

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20 January 2020

issue

3

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20 January 2020

page(s)

263-269

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Osteoimmunology: shared mechanisms and crosstalk between the immune and bone systems

20 January 2020

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AAV-mediated expression of wild-type and ALS-linked mutant VAPB selectively triggers death of motoneurons through a Ca2+-dependent ER-associated pathway

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GM-CSF regulates fusion of mononuclear osteoclasts into bone-resorbing osteoclasts by activating the Ras/ERK pathway.

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PLCgamma2 regulates osteoclastogenesis via its interaction with ITAM proteins and GAB2.

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RANK ligand-induced elevation of cytosolic Ca2+ accelerates nuclear translocation of nuclear factor kappa B in osteoclasts

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A common founder for amyotrophic lateral sclerosis type 8 (ALS8) in the Brazilian population

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