AMP-activated protein kinase inhibits transforming growth factor-beta-induced Smad3-dependent transcription and myofibroblast transdifferentiation. (Q53500309)

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scientific article published on 4 February 2008

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English	AMP-activated protein kinase inhibits transforming growth factor-beta-induced Smad3-dependent transcription and myofibroblast transdifferentiation.	scientific article published on 4 February 2008

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scholarly article

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Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/18250161

AMP-activated protein kinase inhibits transforming growth factor-beta-induced Smad3-dependent transcription and myofibroblast transdifferentiation. (English)

1 reference

Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/18250161

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Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/18250161

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Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/18250161

author name string

Rangnath Mishra

1

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Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/18250161

Barbara L Cool

2

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Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/18250161

Keith R Laderoute

3

1 reference

Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/18250161

Michael S Simonson

6

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Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/18250161

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publication date

4 February 2008

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Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/18250161

number of pages

9

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Journal of Biological Chemistry

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Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/18250161

283

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http://europepmc.org/abstract/MED/18250161

10461-10469

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http://europepmc.org/abstract/MED/18250161

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14 May 2018

http://europepmc.org/abstract/MED/18250161

Myofibroblasts and mechano-regulation of connective tissue remodelling

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The Ca2+/calmodulin-dependent protein kinase kinases are AMP-activated protein kinase kinases

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Short-term overexpression of a constitutively active form of AMP-activated protein kinase in the liver leads to mild hypoglycemia and fatty liver

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AMP-activated protein kinase (AMPK) signaling in endothelial cells is essential for angiogenesis in response to hypoxic stress.

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Concomitant loss of cell surface fibronectin and laminin from transformed rat kidney cells

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Voltage-insensitive Ca2+ channels and Ca2+/calmodulin-dependent protein kinases propagate signals from endothelin-1 receptors to the c-fos promoter

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Hepatocyte growth factor antagonizes the profibrotic action of TGF-beta1 in mesangial cells by stabilizing Smad transcriptional corepressor TGIF

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Pioglitazone attenuates TGF-beta(1)-induction of fibronectin synthesis and its splicing variant in human mesangial cells via activation of peroxisome proliferator-activated receptor (PPAR)gamma

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Identification of a novel AMP-activated protein kinase beta subunit isoform that is highly expressed in skeletal muscle

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Smad3 as a mediator of the fibrotic response

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Immunomodulator FTY720 induces myofibroblast differentiation via the lysophospholipid receptor S1P3 and Smad3 signaling

1 reference

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21 January 2018

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10.1074/JBC.M800902200

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Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/18250161

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Europe PubMed Central

14 May 2018

http://europepmc.org/abstract/MED/18250161

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