Analysis of LKB1 mutations and other molecular alterations in pancreatic acinar cell carcinoma. (Q54373054)

28 May 2018

title

Analysis of LKB1 mutations and other molecular alterations in pancreatic acinar cell carcinoma. (English)

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28 May 2018

author name string

Roeland F de Wilde

1

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28 May 2018

Niki A Ottenhof

2

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28 May 2018

Marnix Jansen

3

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28 May 2018

Folkert H M Morsink

4

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28 May 2018

Wendy W J de Leng

5

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28 May 2018

G Johan A Offerhaus

6

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28 May 2018

Lodewijk A A Brosens

7

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28 May 2018

publication date

13 May 2011

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28 May 2018

published in

Modern Pathology

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28 May 2018

volume

24

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28 May 2018

issue

9

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28 May 2018

page(s)

1229-1236

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28 May 2018

Rare ductal adenocarcinoma of the pancreas in patients younger than age 40 years

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Genetic and immunohistochemical analysis of pancreatic acinar cell carcinoma: frequent allelic loss on chromosome 11p and alterations in the APC/beta-catenin pathway

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Pancreatic tumours: molecular pathways implicated in ductal cancer are involved in ampullary but not in exocrine nonductal or endocrine tumorigenesis

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mTOR inhibitor treatment of pancreatic cancer in a patient With Peutz-Jeghers syndrome

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LKB1 and AMPK family signaling: the intimate link between cell polarity and energy metabolism

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https://api.crossref.org/works/10.1038%2FMODPATHOL.2011.83

Very high risk of cancer in familial Peutz-Jeghers syndrome

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Increased risk of cancer in the Peutz-Jeghers syndrome

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STK11/LKB1 Peutz-Jeghers gene inactivation in intraductal papillary-mucinous neoplasms of the pancreas

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Screening for early pancreatic neoplasia in high-risk individuals: a prospective controlled study.

21 January 2018

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Germline and somatic mutations of the STK11/LKB1 Peutz-Jeghers gene in pancreatic and biliary cancers

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Genetic defects underlying Peutz-Jeghers syndrome (PJS) and exclusion of the polarity-associated MARK/Par1 gene family as potential PJS candidates

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SMAD4 Immunohistochemistry Reflects Genetic Status in Juvenile Polyposis Syndrome

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mTOR and HIF-1alpha-mediated tumor metabolism in an LKB1 mouse model of Peutz-Jeghers syndrome

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Oral rapamycin reduces tumour burden and vascularization in Lkb1(+/-) mice.

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A spontaneous acinar cell carcinoma model for monitoring progression of pancreatic lesions and response to treatment through noninvasive bioluminescence imaging.

21 January 2018

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DAXX/ATRX, MEN1, and mTOR pathway genes are frequently altered in pancreatic neuroendocrine tumors

21 January 2018

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Pancreatic LKB1 deletion leads to acinar polarity defects and cystic neoplasms

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Spontaneous induction of murine pancreatic intraepithelial neoplasia (mPanIN) by acinar cell targeting of oncogenic Kras in adult mice

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Aberrant methylation of CpG islands in intraductal papillary mucinous neoplasms of the pancreas.

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Exocrine pancreatic tumours and their histological classification. A study based on 167 autopsy and 97 surgical cases

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7 January 2021

Acinar cell carcinoma of the pancreas. A clinicopathologic study of 28 cases

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Only wild-type c-Ki-ras codons 12, 13, and 61 in human pancreatic acinar cell carcinomas

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7 January 2021

Evaluation of p53 mutation in pancreatic acinar cell carcinomas of humans and transgenic mice.

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7 January 2021

Allelotype of pancreatic acinar cell carcinoma

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7 January 2021

Clinicopathologic and molecular features of pancreatic adenocarcinoma associated with Peutz-Jeghers syndrome

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7 January 2021

Pancreatic acinar carcinoma shows a distinct pattern of chromosomal imbalances by comparative genomic hybridization

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https://api.crossref.org/works/10.1038%2FMODPATHOL.2011.83

7 January 2021

10.1038/MODPATHOL.2011.83

Identifiers

DOI

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28 May 2018

1 reference

28 May 2018