The p53/p21Cip1/ Waf1 pathway mediates the effects of SPARC on melanoma cell cycle progression. (Q54641862)

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15 February 2020

title

The p53/p21Cip1/ Waf1 pathway mediates the effects of SPARC on melanoma cell cycle progression (English)

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15 February 2020

2

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Mélanie Tichet

3

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15 February 2020

Alexandre Puissant

4

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15 February 2020

Robert Ballotti

9

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15 February 2020

Sophie Tartare-Deckert

10

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15 February 2020

author name string

Maeva Dufies

5

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15 February 2020

Stéphane Rocchi

6

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15 February 2020

Jean-Paul Ortonne

7

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Marcel Deckert

8

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Nina Fenouille

1

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publication date

17 November 2010

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Pigment Cell & Melanoma Research

15 February 2020

published in

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volume

24

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issue

1

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page(s)

219-232

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p21 in cancer: intricate networks and multiple activities

15 February 2020

cites work

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JNK supports survival in melanoma cells by controlling cell cycle arrest and apoptosis.

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Secreted protein acidic and rich in cysteine produced by human melanoma cells modulates polymorphonuclear leukocyte recruitment and antitumor cytotoxic capacity.

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Cathepsin B facilitates autophagy-mediated apoptosis in SPARC overexpressed primitive neuroectodermal tumor cells

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Matricellular proteins: extracellular modulators of cell function

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SPARC, a matricellular protein that functions in cellular differentiation and tissue response to injury

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SPARC, a matricellular protein: at the crossroads of cell-matrix communication

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Enhanced growth of tumors in SPARC null mice is associated with changes in the ECM.

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Requirement for p53 and p21 to sustain G2 arrest after DNA damage

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Mitf cooperates with Rb1 and activates p21Cip1 expression to regulate cell cycle progression

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Mitf regulation of Dia1 controls melanoma proliferation and invasiveness

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A prototypic matricellular protein in the tumor microenvironment--where there's SPARC, there's fire

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WAF1, a potential mediator of p53 tumor suppression

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Cell cycle checkpoints: preventing an identity crisis

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p53 regulation of G(2) checkpoint is retinoblastoma protein dependent

21 January 2018

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Modulation of SPARC expression during butyrate-induced terminal differentiation of cultured human keratinocytes: regulation via a TGF-beta-dependent pathway

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HGF induces fibronectin matrix synthesis in melanoma cells through MAP kinase-dependent signaling pathway and induction of Egr-1.

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Integrative genomic analyses identify MITF as a lineage survival oncogene amplified in malignant melanoma

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Adhesion, migration and communication in melanocytes and melanoma

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Silencing of secreted protein acidic and rich in cysteine inhibits the growth of human melanoma cells with G arrest induction

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Regulation of p53 in response to DNA damage

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p21CIP1 attenuates Ras- and c-Myc-dependent breast tumor epithelial mesenchymal transition and cancer stem cell-like gene expression in vivo.

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SPARC endogenous level, rather than fibroblast-produced SPARC or stroma reorganization induced by SPARC, is responsible for melanoma cell growth

21 January 2018

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SPARC represses E-cadherin and induces mesenchymal transition during melanoma development

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21 January 2018

Identifiers

DOI

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15 February 2020

1 reference