Hemojuvelin: A New Link Between Obesity and Iron Homeostasis (Q57987828)

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Philippe Gual

object named as

Philippe Gual

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Jean Gugenheim

object named as

Jean Gugenheim

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author name string

Nathalie Luciani

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Carole Brasse-Lagnel

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Maura Poli

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Celine Lesueur

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Annie Laquerriere

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Vanessa Folope

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Yannick LeMarchand-Brustel

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Albert Tran

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Soumeya Bekri

Increased adipose tissue expression of hepcidin in severe obesity is independent from diabetes and NASH

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publication date

10 February 2011

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published in

Obesity

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volume

19

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issue

8

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page(s)

1545-1551

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cites work

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Bone morphogenetic proteins 2, 4, and 9 stimulate murine hepcidin 1 expression independently of Hfe, transferrin receptor 2 (Tfr2), and IL-6

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Modulation of bone morphogenetic protein signaling in vivo regulates systemic iron balance

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Soluble hemojuvelin is released by proprotein convertase-mediated cleavage at a conserved polybasic RNRR site

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BMP6 is a key endogenous regulator of hepcidin expression and iron metabolism

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Hemojuvelin regulates hepcidin expression via a selective subset of BMP ligands and receptors independently of neogenin

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The inflammatory C-reactive protein is increased in both liver and adipose tissue in severely obese patients independently from metabolic syndrome, Type 2 diabetes, and NASH

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