Tumor necrosis factor-alpha inhibits pre-osteoblast differentiation through its type-1 receptor (Q73879531)

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7 November 2019

title

Tumor necrosis factor-alpha inhibits pre-osteoblast differentiation through its type-1 receptor (English)

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7 November 2019

3

John C Clohisy

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7 November 2019

author name string

Sabiha Abbas

series ordinal

1

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7 November 2019

Yan-Hong Zhang

series ordinal

2

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7 November 2019

Yousef Abu-Amer

series ordinal

4

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7 November 2019

publication date

1 April 2003

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7 November 2019

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7 November 2019

volume

22

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7 November 2019

issue

1-2

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7 November 2019

page(s)

33-41

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Tumor necrosis factor: a pleiotropic cytokine and therapeutic target

7 November 2019

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Tumor necrosis factor-α mediates polymethylmethacrylate particle-induced NF-κB activation in osteoclast precursor cells

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Tumor necrosis factor-alpha mediates orthopedic implant osteolysis.

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Production of cytokines around loosened cemented acetabular components. Analysis with immunohistochemical techniques and in situ hybridization

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On the role of tumor necrosis factor and receptors in models of multiorgan failure, rheumatoid arthritis, multiple sclerosis and inflammatory bowel disease

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Involvement of receptor activator of NFkappaB ligand and tumor necrosis factor-alpha in bone destruction in rheumatoid arthritis

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Inhibition of Osteoblast Differentiation by Tumor Necrosis Factor-

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Effects of cytokines on alkaline phosphatase and osteocalcin production, calcification and calcium release by human osteoblastic cells

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Effects of interleukin-1 beta and tumor necrosis factor-alpha on osteoblastic expression of osteocalcin and mineralized extracellular matrix in vitro

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Lipopolysaccharide-stimulated osteoclastogenesis is mediated by tumor necrosis factor via its P55 receptor.

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TRADD-TRAF2 and TRADD-FADD interactions define two distinct TNF receptor 1 signal transduction pathways

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The TRAF family of signal transducers mediates NF-kappaB activation by the TRANCE receptor

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Tumor necrosis factor-alpha induces differentiation of and bone resorption by osteoclasts

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Tumor necrosis factor-alpha supports the survival of osteoclasts through the activation of Akt and ERK.

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Tumor necrosis factor-alpha (TNF) stimulates RANKL-induced osteoclastogenesis via coupling of TNF type 1 receptor and RANK signaling pathways

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7 January 2021

Marrow stromal stem cells

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Multilineage gene expression precedes commitment in the hemopoietic system

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Multilineage potential of adult human mesenchymal stem cells

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Osf2/Cbfa1: a transcriptional activator of osteoblast differentiation

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7 January 2021

Targeted disruption of Cbfa1 results in a complete lack of bone formation owing to maturational arrest of osteoblasts

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runt homology domain transcription factors (Runx, Cbfa, and AML) mediate repression of the bone sialoprotein promoter: evidence for promoter context-dependent activity of Cbfa proteins

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7 January 2021

Tumor necrosis factor in the pathogenesis of infectious diseases

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

Transcriptional roles of nuclear factor kappa B and nuclear factor-interleukin-6 in the tumor necrosis factor alpha-dependent induction of cyclooxygenase-2 in MC3T3-E1 cells

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7 January 2021

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Identifiers

DOI

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7 November 2019

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