Activation of STAT6 by STING is critical for antiviral innate immunity (Q24293312)

Interferon regulatory factor 3

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identical protein binding

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TANK-binding kinase 1

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Transmembrane protein 173

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Huihui Chen

1

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Hui Sun

2

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Fuping You

3

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Wenxiang Sun

4

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Xiang Zhou

5

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Lu Chen

6

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Jing Yang

7

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Yutao Wang

8

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Hong Tang

9

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Yukun Guan

10

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Weiwei Xia

11

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Jun Gu

12

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Hiroki Ishikawa

13

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Delia Gutman

14

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Glen Barber

15

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Zhihai Qin

16

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Zhengfan Jiang

Membrane-bound eotaxin-3 mediates eosinophil transepithelial migration in IL-4-stimulated epithelial cells

17

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language of work or name

English

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publication date

14 October 2011

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published in

Cell

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volume

147

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issue

2

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page(s)

436-46

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Recognition of cytosolic DNA activates an IRF3-dependent innate immune response

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The adaptor protein MITA links virus-sensing receptors to IRF3 transcription factor activation

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Discrete roles of STAT4 and STAT6 transcription factors in tuning epigenetic modifications and transcription during T helper cell differentiation

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Lack of IL-4-induced Th2 response and IgE class switching in mice with disrupted Stat6 gene

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The DEAD-box helicase DDX3X is a critical component of the TANK-binding kinase 1-dependent innate immune response

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STING is an endoplasmic reticulum adaptor that facilitates innate immune signalling

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STING regulates intracellular DNA-mediated, type I interferon-dependent innate immunity

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CD14 is required for MyD88-independent LPS signaling.

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A Toll-like receptor-independent antiviral response induced by double-stranded B-form DNA.

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RIG-I-dependent sensing of poly(dA:dT) through the induction of an RNA polymerase III-transcribed RNA intermediate

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PCBP2 mediates degradation of the adaptor MAVS via the HECT ubiquitin ligase AIP4

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Defective TNF-alpha-induced apoptosis in STAT1-null cells due to low constitutive levels of caspases

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