Paclitaxel-induced apoptosis in BJAB cells proceeds via a death receptor-independent, caspases-3/-8-driven mitochondrial amplification loop (Q33186742)

23 July 2017

title

Paclitaxel-induced apoptosis in BJAB cells proceeds via a death receptor-independent, caspases-3/-8-driven mitochondrial amplification loop (English)

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23 July 2017

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1

Clarissa von Haefen

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23 July 2017

Thomas Wieder

2

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23 July 2017

Frank Essmann

3

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23 July 2017

Klaus Schulze-Osthoff

4

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23 July 2017

Bernd Dörken

5

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23 July 2017

Peter T Daniel

6

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23 July 2017

language of work or name

English

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publication date

1 April 2003

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23 July 2017

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23 July 2017

volume

22

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23 July 2017

issue

15

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23 July 2017

page(s)

2236-2247

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Inhibition of drug-induced Fas ligand transcription and apoptosis by Bcl-XL.

23 July 2017

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Overexpression of caspase-3 restores sensitivity for drug-induced apoptosis in breast cancer cell lines with acquired drug resistance

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Sensitization for death receptor- or drug-induced apoptosis by re-expression of caspase-8 through demethylation or gene transfer

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Cell type specific involvement of death receptor and mitochondrial pathways in drug-induced apoptosis

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Inhibition of human caspases by peptide-based and macromolecular inhibitors

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Adenovirus-mediated overexpression of p14(ARF) induces p53 and Bax-independent apoptosis

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Death receptor recruitment of endogenous caspase-10 and apoptosis initiation in the absence of caspase-8

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Involvement of CD95-independent caspase 8 activation in arsenic trioxide-induced apoptosis

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Pro-apoptotic cascade activates BID, which oligomerizes BAK or BAX into pores that result in the release of cytochrome c.

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Rapid kinetics of tBid-induced cytochrome c and Smac/DIABLO release and mitochondrial depolarization

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FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

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2-Chloro-2'-deoxyadenosine induces apoptosis through the Fas/Fas ligand pathway in human leukemia cell line MOLT-4.

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A crucial role of caspase 3 and caspase 8 in paclitaxel-induced apoptosis

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Relapse in childhood acute lymphoblastic leukemia is associated with a decrease of the Bax/Bcl-2 ratio and loss of spontaneous caspase-3 processing in vivo

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The apoptosis promoting Bcl-2 homologues Bak and Nbk/Bik overcome drug resistance in Mdr-1-negative and Mdr-1-overexpressing breast cancer cell lines

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Bcl-2 oncoprotein protects the human prostatic carcinoma cell line PC3 from TRAIL-mediated apoptosis.

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Ceramide induces mitochondrial activation and apoptosis via a Bax-dependent pathway in human carcinoma cells

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The effect of antimicrotubule agents on signal transduction pathways of apoptosis: a review

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Activation of caspase-8 in drug-induced apoptosis of B-lymphoid cells is independent of CD95/Fas receptor-ligand interaction and occurs downstream of caspase-3.

21 January 2018

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Bid acts on the permeability transition pore complex to induce apoptosis

21 January 2018

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Target protease specificity of the viral serpin CrmA. Analysis of five caspases

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7 January 2021

Sensitization of resistant lymphoma cells to irradiation-induced apoptosis by the death ligand TRAIL

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7 January 2021

Caspase-8/FLICE functions as an executioner caspase in anticancer drug-induced apoptosis

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7 January 2021

Serine protease inhibitor TPCK prevents Taxol-induced cell death and blocks c-Raf-1 and Bcl-2 phosphorylation in human breast carcinoma cells

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7 January 2021

Cytochrome c release is upstream to activation of caspase-9, caspase-8, and caspase-3 in the enhanced apoptosis of anaplastic thyroid cancer cells induced by manumycin and paclitaxel

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7 January 2021

Identifiers

DOI

10.1038/SJ.ONC.1206280

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Dimensions Publication ID

23 July 2017

1024736065

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