Cooperative signaling via transcription factors NF-κB and AP1/c-Fos mediates endothelial cell STIM1 expression and hyperpermeability in response to endotoxin (Q34107201)

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English	Cooperative signaling via transcription factors NF-κB and AP1/c-Fos mediates endothelial cell STIM1 expression and hyperpermeability in response to endotoxin	scientific article

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Europe PubMed Central

PubMed publication ID

31 July 2017

Cooperative signaling via transcription factors NF-κB and AP1/c-Fos mediates endothelial cell STIM1 expression and hyperpermeability in response to endotoxin (English)

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Europe PubMed Central

PubMed publication ID

31 July 2017

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12 June 2020

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Auditi DebRoy

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12 June 2020

Stephen M Vogel

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12 June 2020

Premanand C Sundivakkam

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12 June 2020

Asrar B Malik

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12 June 2020

Chinnaswamy Tiruppathi

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12 June 2020

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11 July 2014

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31 July 2017

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14

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Journal of Biological Chemistry

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12 June 2020

289

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12 June 2020

35

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12 June 2020

24188-24201

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12 June 2020

The transcription factor DREAM represses the deubiquitinase A20 and mediates inflammation.

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5 July 2018

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Store-operated Ca2+ entry (SOCE) induced by protease-activated receptor-1 mediates STIM1 protein phosphorylation to inhibit SOCE in endothelial cells through AMP-activated protein kinase and p38β mitogen-activated protein kinase

1 reference

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STIM1 controls endothelial barrier function independently of Orai1 and Ca2+ entry

1 reference

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TLR4 activation of TRPC6-dependent calcium signaling mediates endotoxin-induced lung vascular permeability and inflammation

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Regulation of NF-κB signaling by the A20 deubiquitinase

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The Ca(2+) sensor stromal interaction molecule 1 (STIM1) is necessary and sufficient for the store-operated Ca(2+) entry function of transient receptor potential canonical (TRPC) 1 and 4 channels in endothelial cells

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5 July 2018

Transcription factor NF-κB regulates expression of pore-forming Ca2+ channel unit, Orai1, and its activator, STIM1, to control Ca2+ entry and affect cellular functions

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5 July 2018

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S-glutathionylation activates STIM1 and alters mitochondrial homeostasis.

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5 July 2018

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STIM1 gates TRPC channels, but not Orai1, by electrostatic interaction

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Ca2+ influx induced by protease-activated receptor-1 activates a feed-forward mechanism of TRPC1 expression via nuclear factor-kappaB activation in endothelial cells

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Impairment of store-operated Ca2+ entry in TRPC4(-/-) mice interferes with increase in lung microvascular permeability

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Systemic activation of tissue-factor dependent coagulation pathway in evolving acute respiratory distress syndrome in patients with trauma and sepsis

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10.1074/JBC.M114.570051

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25016017%20AND%20SRC:MED&resulttype=core&format=json

12 June 2020

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12 June 2020

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12 June 2020

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