Highly specific ubiquitin-competing molecules effectively promote frataxin accumulation and partially rescue the aconitase defect in Friedreich ataxia cells. (Q35174542)
Jump to navigation
Jump to search
scientific article
Language | Label | Description | Also known as |
---|---|---|---|
English | Highly specific ubiquitin-competing molecules effectively promote frataxin accumulation and partially rescue the aconitase defect in Friedreich ataxia cells. |
scientific article |
Statements
Highly specific ubiquitin-competing molecules effectively promote frataxin accumulation and partially rescue the aconitase defect in Friedreich ataxia cells (English)
Alessandra Rufini
Silvia Fortuni
Gabriella De Martino
Ottaviano Incani
Almerinda Di Venere
Monica Benini
Damiano Sergio Massaro
Gaetano Arcuri
Dario Serio
Roberto Testi
27 December 2014
1 reference
1 reference