Amyloid-beta(1-42) fibrillar precursors are optimal for inducing tumor necrosis factor-alpha production in the THP-1 human monocytic cell line (Q37358028)

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scientific article published on September 2009

Language	Label	Description	Also known as
English	Amyloid-beta(1-42) fibrillar precursors are optimal for inducing tumor necrosis factor-alpha production in the THP-1 human monocytic cell line	scientific article published on September 2009

Statements

scholarly article

1 reference

Europe PubMed Central

PMC publication ID

19 August 2017

Amyloid-beta(1-42) fibrillar precursors are optimal for inducing tumor necrosis factor-alpha production in the THP-1 human monocytic cell line (English)

1 reference

Europe PubMed Central

PMC publication ID

19 August 2017

author name string

Deepa Ajit

1

1 reference

Europe PubMed Central

PMC publication ID

19 August 2017

Maria L D Udan

2

1 reference

Europe PubMed Central

PMC publication ID

19 August 2017

Geeta Paranjape

3

1 reference

Europe PubMed Central

PMC publication ID

19 August 2017

Michael R Nichols

4

1 reference

Europe PubMed Central

PMC publication ID

19 August 2017

language of work or name

0 references

publication date

1 September 2009

1 reference

Europe PubMed Central

PMC publication ID

19 August 2017

1 reference

Europe PubMed Central

PMC publication ID

19 August 2017

48

1 reference

Europe PubMed Central

PMC publication ID

19 August 2017

38

1 reference

Europe PubMed Central

PMC publication ID

19 August 2017

9011-9021

1 reference

Europe PubMed Central

PMC publication ID

19 August 2017

Oligomeric amyloid beta associates with postsynaptic densities and correlates with excitatory synapse loss near senile plaques

1 reference

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6 September 2017

Rapid appearance and local toxicity of amyloid-beta plaques in a mouse model of Alzheimer's disease

1 reference

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6 September 2017

Fibril specific, conformation dependent antibodies recognize a generic epitope common to amyloid fibrils and fibrillar oligomers that is absent in prefibrillar oligomers

1 reference

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6 September 2017

Intracellular amyloid-beta in Alzheimer's disease

1 reference

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6 September 2017

Soluble protein oligomers in neurodegeneration: lessons from the Alzheimer's amyloid beta-peptide

1 reference

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6 September 2017

Self-propagating, molecular-level polymorphism in Alzheimer's beta-amyloid fibrils

1 reference

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6 September 2017

Cell biology of protein misfolding: the examples of Alzheimer's and Parkinson's diseases

1 reference

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ULTRASTRUCTURAL STUDIES IN ALZHEIMER'S PRESENILE DEMENTIA

1 reference

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Intrathecal inflammation precedes development of Alzheimer's disease

1 reference

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6 September 2017

Common structure of soluble amyloid oligomers implies common mechanism of pathogenesis

1 reference

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In vitro characterization of conditions for amyloid-beta peptide oligomerization and fibrillogenesis

1 reference

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Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-term potentiation in vivo

1 reference

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Conformational Abs recognizing a generic amyloid fibril epitope

1 reference

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6 September 2017

beta-Amyloid stimulation of microglia and monocytes results in TNFalpha-dependent expression of inducible nitric oxide synthase and neuronal apoptosis.

1 reference

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6 September 2017

The cell biology of beta-amyloid precursor protein and presenilin in Alzheimer's disease

1 reference

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6 September 2017

RAGE and amyloid-beta peptide neurotoxicity in Alzheimer's disease

1 reference

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6 September 2017

Beta-amyloid neurotoxicity requires fibril formation and is inhibited by congo red.

1 reference

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6 September 2017

Structural model for the β-amyloid fibril based on interstrand alignment of an antiparallel-sheet comprising a C-terminal peptide

1 reference

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Evaluation of a soluble tetrazolium/formazan assay for cell growth and drug sensitivity in culture using human and other tumor cell lines

1 reference

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6 September 2017

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

1 reference

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6 September 2017

Toll-like receptors 2 and 4 mediate Abeta(1-42) activation of the innate immune response in a human monocytic cell line

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2749082

14 September 2017

Role of the toll-like receptor 4 in neuroinflammation in Alzheimer's disease.

1 reference

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14 September 2017

A cell surface receptor complex for fibrillar beta-amyloid mediates microglial activation.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2749082

14 September 2017

Amyloid beta and amylin fibrils induce increases in proinflammatory cytokine and chemokine production by THP-1 cells and murine microglia

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2749082

28 September 2017

Interaction of Alzheimer beta-amyloid peptide with the human monocytic cell line THP-1 results in a protein kinase C-dependent secretion of tumor necrosis factor-alpha

1 reference

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Scavenger receptor-mediated adhesion of microglia to beta-amyloid fibrils

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2749082

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Seeding "one-dimensional crystallization" of amyloid: a pathogenic mechanism in Alzheimer's disease and scrapie?

1 reference

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Microglia and cytokines in neurological disease, with special reference to AIDS and Alzheimer's disease

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beta-Amyloid induces increased release of interleukin-1 beta from lipopolysaccharide-activated human monocytes

1 reference

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28 September 2017

Inflammation takes on Alzheimer disease.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2749082

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Activation of microglial cells by beta-amyloid protein and interferon-gamma.

1 reference

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Reactive microglia in patients with senile dementia of the Alzheimer type are positive for the histocompatibility glycoprotein HLA-DR.

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In vitro aging of beta-amyloid protein causes peptide aggregation and neurotoxicity

1 reference

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Abeta peptide conformation determines uptake and interleukin-1alpha expression by primary microglial cells

1 reference

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Different conformations of amyloid beta induce neurotoxicity by distinct mechanisms in human cortical neurons.

1 reference

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Targeting amyloid-beta peptide (Abeta) oligomers by passive immunization with a conformation-selective monoclonal antibody improves learning and memory in Abeta precursor protein (APP) transgenic mice.

1 reference

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In vitro preparation of prefibrillar intermediates of amyloid-beta and alpha-synuclein.

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Amyloid-beta protofibrils differ from amyloid-beta aggregates induced in dilute hexafluoroisopropanol in stability and morphology.

1 reference

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Solid state NMR reveals a pH-dependent antiparallel beta-sheet registry in fibrils formed by a beta-amyloid peptide

1 reference

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The LPS receptor (CD14) links innate immunity with Alzheimer's disease

1 reference

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Oligomeric and fibrillar species of amyloid-beta peptides differentially affect neuronal viability.

1 reference

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Growth of beta-amyloid(1-40) protofibrils by monomer elongation and lateral association. Characterization of distinct products by light scattering and atomic force microscopy.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2749082

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Imaging of amyloid-beta deposits in brains of living mice permits direct observation of clearance of plaques with immunotherapy.

1 reference

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Assembly of A beta amyloid protofibrils: an in vitro model for a possible early event in Alzheimer's disease

1 reference

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The importance of inflammatory mechanisms in Alzheimer disease.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2749082

2 September 2018

Amyloid beta-protein fibrillogenesis. Detection of a protofibrillar intermediate.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2749082

2 September 2018

Insights into the amyloid folding problem from solid-state NMR

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2 October 2018

Amyloid fibril formation by A beta 16-22, a seven-residue fragment of the Alzheimer's beta-amyloid peptide, and structural characterization by solid state NMR

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2 October 2018

Amyloid beta-protein fibrillogenesis. Structure and biological activity of protofibrillar intermediates

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12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Observation of metastable Abeta amyloid protofibrils by atomic force microscopy

1 reference

https://pubmed.ncbi.nlm.nih.gov/19694428

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Inflammatory factors are elevated in brain microvessels in Alzheimer's disease

1 reference

https://pubmed.ncbi.nlm.nih.gov/19694428

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1021/BI9003777

1 reference

Europe PubMed Central

PMC publication ID

19 August 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

19 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

19 August 2017

ResearchGate publication ID

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