Trientine reduces BACE1 activity and mitigates amyloidosis via the AGE/RAGE/NF-κB pathway in a transgenic mouse model of Alzheimer's disease. (Q37406597)

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scientific article published on 03 May 2013

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English	Trientine reduces BACE1 activity and mitigates amyloidosis via the AGE/RAGE/NF-κB pathway in a transgenic mouse model of Alzheimer's disease.	scientific article published on 03 May 2013

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scholarly article

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27 February 2020

Trientine reduces BACE1 activity and mitigates amyloidosis via the AGE/RAGE/NF-κB pathway in a transgenic mouse model of Alzheimer's disease (English)

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27 February 2020

Alzheimer's disease

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3

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27 February 2020

author name string

Chun-Yan Wang

1

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27 February 2020

Jing-Wei Xie

2

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27 February 2020

Tao Wang

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27 February 2020

Jian-Hui Cai

5

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27 February 2020

Xu Wang

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27 February 2020

Bao-Lu Zhao

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27 February 2020

Li An

8

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27 February 2020

Zhan-You Wang

9

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27 February 2020

publication date

3 May 2013

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27 February 2020

Antioxidants & Redox Signaling

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27 February 2020

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27 February 2020

2024-2039

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27 February 2020

17

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27 February 2020

Copper and oxidative stress in the pathogenesis of Alzheimer's disease

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Therapeutic potential of copper chelation with triethylenetetramine in managing diabetes mellitus and Alzheimer's disease

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Dysregulated NF-κB pathway in peripheral mononuclear cells of Alzheimer's disease patients

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Huperzine A activates Wnt/β-catenin signaling and enhances the nonamyloidogenic pathway in an Alzheimer transgenic mouse model

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The beta-secretase enzyme BACE in health and Alzheimer's disease: regulation, cell biology, function, and therapeutic potential

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RAGE regulates BACE1 and Abeta generation via NFAT1 activation in Alzheimer's disease animal model.

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Hippocampal RAGE immunoreactivity in early and advanced Alzheimer's disease

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Amyloid-beta protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory

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Central insulin resistance as a trigger for sporadic Alzheimer-like pathology: an experimental approach

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Alzheimer's disease is a synaptic failure

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RAGE is a multiligand receptor of the immunoglobulin superfamily: implications for homeostasis and chronic disease

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The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

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Lipid peroxidation and protein oxidation in Alzheimer's disease brain: potential causes and consequences involving amyloid beta-peptide-associated free radical oxidative stress

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Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-term potentiation in vivo

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Treatment with a copper-zinc chelator markedly and rapidly inhibits beta-amyloid accumulation in Alzheimer's disease transgenic mice.

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AGEs/RAGE complex upregulates BACE1 via NF-κB pathway activation.

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Identifiers

10.1089/ARS.2012.5158

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Europe PubMed Central

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27 February 2020

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Europe PubMed Central

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27 February 2020

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27 February 2020

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