p53 deficiency provokes urothelial proliferation and synergizes with activated Ha-ras in promoting urothelial tumorigenesis. (Q38345714)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:14737103%20AND%20SRC:MED&resulttype=core&format=json

17 November 2019

title

p53 deficiency provokes urothelial proliferation and synergizes with activated Ha-ras in promoting urothelial tumorigenesis (English)

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17 November 2019

7

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17 November 2019

Tung-Tien Sun

8

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17 November 2019

author name string

Jing Gao

1

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17 November 2019

Hong-Ying Huang

2

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17 November 2019

Joanne Pak

3

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17 November 2019

Jin Cheng

4

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17 November 2019

Zhong-Ting Zhang

5

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17 November 2019

Ellen Shapiro

6

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17 November 2019

Xue-Ru Wu

9

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17 November 2019

language of work or name

English

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publication date

1 January 2004

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17 November 2019

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17 November 2019

volume

23

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17 November 2019

issue

3

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17 November 2019

page(s)

687-696

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1207169

17 November 2019

exact match

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Regulation of p53 after different forms of stress and at different cell cycle stages

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A tissue-specific promoter that can drive a foreign gene to express in the suprabasal urothelial cells of transgenic mice.

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The C-terminal domain of p53 recognizes DNA damaged by ionizing radiation

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A mutant p53 protein is required for maintenance of the transformed phenotype in cells transformed with p53 plus ras cDNAs

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Tissue-specific inactivation of p53 tumor suppression in the mouse.

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Concurrent mutations of coding and regulatory sequences of the Ha-ras gene in urinary bladder carcinomas

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Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumours

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Oncogenic ras and p53 cooperate to induce cellular senescence

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Identification of H-ras mutations in urine sediments complements cytology in the detection of bladder tumors

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Activation of c-myc gene expression by tumor-derived p53 mutants requires a discrete C-terminal domain

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7 January 2021

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Overexpression of p53 protein in a high-risk population of patients with superficial bladder cancer before and after bacillus Calmette-Guérin therapy: correlation to clinical outcome

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'Gain of function' phenotype of tumor-derived mutant p53 requires the oligomerization/nonsequence-specific nucleic acid-binding domain

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p53 Expression Predicts Progression and Poor Survival in T1 Bladder Tumours

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7 January 2021

Growth factors and bladder cancer: clinical implications of the interactions between growth factors and their urothelial receptors

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p53 and microvessel density in primary resection specimens of superficial bladder cancer

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7 January 2021

Review of chromosome studies in urological tumors. II. Cytogenetics and molecular genetics of bladder cancer.

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Association of P53 nuclear overexpression and tumor progression in carcinoma in situ of the bladder.

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7 January 2021

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7 January 2021

Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

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7 January 2021

Identification of a minimal transforming domain of p53: negative dominance through abrogation of sequence-specific DNA binding

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7 January 2021

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Role of Ha-ras activation in superficial papillary pathway of urothelial tumor formation

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Identifiers

DOI

10.1038/SJ.ONC.1207169

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:14737103%20AND%20SRC:MED&resulttype=core&format=json

Dimensions Publication ID

17 November 2019

0 references

1 reference