Melanoma tumors frequently acquire LRP2/megalin expression, which modulates melanoma cell proliferation and survival rates. (Q38920389)

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27 February 2020

title

Melanoma tumors frequently acquire LRP2/megalin expression, which modulates melanoma cell proliferation and survival rates (English)

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27 February 2020

6

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27 February 2020

Mai-Britt H Thomsen

7

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27 February 2020

author name string

Rikke K Andersen

1

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27 February 2020

Katrine Hammer

2

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27 February 2020

Henrik Hager

3

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27 February 2020

Julie N Christensen

4

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27 February 2020

Maja Ludvigsen

5

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27 February 2020

Mette Madsen

8

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27 February 2020

publication date

13 February 2015

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Pigment Cell & Melanoma Research

27 February 2020

published in

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27 February 2020

volume

28

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27 February 2020

issue

3

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27 February 2020

page(s)

267-280

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Clusterin activates survival through the phosphatidylinositol 3-kinase/Akt pathway

27 February 2020

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1,25-Dihydroxyvitamin D₃ enhances NK susceptibility of human melanoma cells via Hsp60-mediated FAS expression

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Targeting GRP78 to enhance melanoma cell death.

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The RAS/RAF/MEK/ERK and PI3K/AKT signaling pathways present molecular targets for the effective treatment of advanced melanoma

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Targeting hyperactivation of the AKT survival pathway to overcome therapy resistance of melanoma brain metastases

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Megalin-dependent yellow endocytosis restricts melanization in the Drosophila cuticle

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Comprehensive expression profiling of tumor cell lines identifies molecular signatures of melanoma progression

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EMT-activating transcription factors in cancer: beyond EMT and tumor invasiveness.

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Ras, PI(3)K and mTOR signalling controls tumour cell growth

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Renal phenotypic investigations of megalin-deficient patients: novel insights into tubular proteinuria and albumin filtration.

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Megalin-deficiency causes high myopia, retinal pigment epithelium-macromelanosomes and abnormal development of the ciliary body in mice

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21 January 2018

Identifiers

DOI

10.1111/PCMR.12352

1 reference

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27 February 2020

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