Regulation of HAX-1 anti-apoptotic protein by Omi/HtrA2 protease during cell death (Q40515224)

21 September 2017

title

Regulation of HAX-1 anti-apoptotic protein by Omi/HtrA2 protease during cell death (English)

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21 September 2017

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Lucia Cilenti

1

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21 September 2017

Mangala M Soundarapandian

2

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21 September 2017

George A Kyriazis

3

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21 September 2017

Valerie Stratico

4

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21 September 2017

Supriya Singh

5

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21 September 2017

Sanjeev Gupta

6

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21 September 2017

Joseph V Bonventre

7

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21 September 2017

Emad S Alnemri

8

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21 September 2017

Antonis S Zervos

9

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21 September 2017

language of work or name

English

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publication date

15 September 2004

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Journal of Biological Chemistry

21 September 2017

published in

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21 September 2017

volume

279

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21 September 2017

issue

48

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21 September 2017

page(s)

50295-50301

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A serine protease, HtrA2, is released from the mitochondria and interacts with XIAP, inducing cell death

21 September 2017

cites work

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https://api.crossref.org/works/10.1074%2FJBC.M406006200

Identification of Omi/HtrA2 as a mitochondrial apoptotic serine protease that disrupts inhibitor of apoptosis protein-caspase interaction

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

The serine protease Omi/HtrA2 regulates apoptosis by binding XIAP through a reaper-like motif

21 January 2018

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https://api.crossref.org/works/10.1074%2FJBC.M406006200

HtrA2 promotes cell death through its serine protease activity and its ability to antagonize inhibitor of apoptosis proteins

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

Omi/HtrA2 catalytic cleavage of inhibitor of apoptosis (IAP) irreversibly inactivates IAPs and facilitates caspase activity in apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

Inhibitor of apoptosis proteins are substrates for the mitochondrial serine protease Omi/HtrA2

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

Intramitochondrial serine protease activity of Omi/HtrA2 is required for caspase-independent cell death of human neutrophils

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

Mitochondrial protease Omi/HtrA2 enhances caspase activation through multiple pathways

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

Loss of Omi mitochondrial protease activity causes the neuromuscular disorder of mnd2 mutant mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

Mxi1, a protein that specifically interacts with Max to bind Myc-Max recognition sites

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

Cdi1, a human G1 and S phase protein phosphatase that associates with Cdk2

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

Characterization of a novel and specific inhibitor for the pro-apoptotic protease Omi/HtrA2.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

The C-terminal tail of presenilin regulates Omi/HtrA2 protease activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

Three-color flow cytometric assay for the study of the mechanisms of cell-mediated cytotoxicity.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

A rapid single-laser flow cytometric method for discrimination of early apoptotic cells in a heterogenous cell population.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

F16, a mitochondriotoxic compound, triggers apoptosis or necrosis depending on the genetic background of the target carcinoma cell

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

A flow cytometry assay simultaneously detects independent apoptotic parameters

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

mnd2: a new mouse model of inherited motor neuron disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

The polycystic kidney disease protein PKD2 interacts with Hax-1, a protein associated with the actin cytoskeleton

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

K15 protein of Kaposi's sarcoma-associated herpesvirus is latently expressed and binds to HAX-1, a protein with antiapoptotic function

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

Characterization of a novel human serine protease that has extensive homology to bacterial heat shock endoprotease HtrA and is regulated by kidney ischemia

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

A temperature-dependent switch from chaperone to protease in a widely conserved heat shock protein.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

Crystal structure of DegP (HtrA) reveals a new protease-chaperone machine

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

HtrA2/Omi, a sheep in wolf's clothing

21 January 2018

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https://api.crossref.org/works/10.1074%2FJBC.M406006200

Programmed cell death: alive and well in the new millennium

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

Epstein-Barr virus (EBV) nuclear antigen leader protein (EBNA-LP) forms complexes with a cellular anti-apoptosis protein Bcl-2 or its EBV counterpart BHRF1 through HS1-associated protein X-1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

Epstein-Barr virus nuclear antigen 5 interacts with HAX-1, a possible component of the B-cell receptor signalling pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

HAX-1, identified by differential display reverse transcription polymerase chain reaction, is overexpressed in lesional psoriasis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

Omi/HtrA2 promotes cell death by binding and degrading the anti-apoptotic protein ped/pea-15

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M406006200

21 January 2018

Identifiers

DOI

10.1074/JBC.M406006200

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21 September 2017

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