Non-apoptotic role of BID in inflammation and innate immunity. (Q50521157)

14 March 2018

title

Non-apoptotic role of BID in inflammation and innate immunity. (English)

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14 March 2018

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Ricardo G Correa

2

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Douglas R. Green

object named as

Douglas R Green

6

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author name string

Garabet Yeretssian

1

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14 March 2018

Karine Doiron

3

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14 March 2018

Patrick Fitzgerald

4

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Christopher P Dillon

5

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John C Reed

7

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Maya Saleh

8

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language of work or name

English

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publication date

8 May 2011

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14 March 2018

published in

Nature

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14 March 2018

volume

474

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14 March 2018

issue

7349

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14 March 2018

page(s)

96-99

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14 March 2018

Homeostasis and inflammation in the intestine

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Nod1 detects a unique muropeptide from gram-negative bacterial peptidoglycan

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Nod1 acts as an intracellular receptor to stimulate chemokine production and neutrophil recruitment in vivo

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Reciprocal cross-talk between Nod2 and TAK1 signaling pathways

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Nod2-dependent regulation of innate and adaptive immunity in the intestinal tract

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Human CARD4 protein is a novel CED-4/Apaf-1 cell death family member that activates NF-kappaB

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Reconstituted NALP1 inflammasome reveals two-step mechanism of caspase-1 activation

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Bcl-2 and Bcl-XL regulate proinflammatory caspase-1 activation by interaction with NALP1

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The C. elegans protein EGL-1 is required for programmed cell death and interacts with the Bcl-2-like protein CED-9

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Phosphorylation of bid by casein kinases I and II regulates its cleavage by caspase 8.

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Unravelling the pathogenesis of inflammatory bowel disease

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Bid mediates anti-apoptotic COX-2 induction through the IKKbeta/NFkappaB pathway due to 5-MCDE exposure

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BID: a novel BH3 domain-only death agonist

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Mechanism of Bcl-2 and Bcl-X(L) inhibition of NLRP1 inflammasome: loop domain-dependent suppression of ATP binding and oligomerization

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The Crohn's disease protein, NOD2, requires RIP2 in order to induce ubiquitinylation of a novel site on NEMO

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The adaptor protein CARD9 is required for innate immune responses to intracellular pathogens

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Cellular inhibitors of apoptosis cIAP1 and cIAP2 are required for innate immunity signaling by the pattern recognition receptors NOD1 and NOD2.

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Nod2, a Nod1/Apaf-1 family member that is restricted to monocytes and activates NF-kappaB

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A Simple Statistical Parameter for Use in Evaluation and Validation of High Throughput Screening Assays

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The PANTHER database of protein families, subfamilies, functions and pathways

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Enrichment or depletion of a GO category within a class of genes: which test?

21 January 2018

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Cluster analysis and display of genome-wide expression patterns

21 January 2018

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Java Treeview--extensible visualization of microarray data

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Humanin binds and nullifies Bid activity by blocking its activation of Bax and Bak

21 January 2018

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Muramyl dipeptide activation of nucleotide-binding oligomerization domain 2 protects mice from experimental colitis.

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PubMed

https://pubmed.ncbi.nlm.nih.gov/21552281

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Activation of toll-like receptor 3 protects against DSS-induced acute colitis

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PubMed

https://pubmed.ncbi.nlm.nih.gov/21552281

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1038/NATURE09982

1 reference

14 March 2018

Dimensions Publication ID

1052539206

0 references

1 reference

14 March 2018