Mutation of ERBB2 provides a novel alternative mechanism for the ubiquitous activation of RAS-MAPK in ovarian serous low malignant potential tumors. (Q50613227)

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scientific article published in November 2008

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English	Mutation of ERBB2 provides a novel alternative mechanism for the ubiquitous activation of RAS-MAPK in ovarian serous low malignant potential tumors.	scientific article published in November 2008

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16 March 2018

http://europepmc.org/abstract/MED/19010816

Mutation of ERBB2 provides a novel alternative mechanism for the ubiquitous activation of RAS-MAPK in ovarian serous low malignant potential tumors (English)

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Erb-b2 receptor tyrosine kinase 2

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Richard W. Tothill

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Michael S Anglesio

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Nicola J Waddell

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Georgia Chenevix-Trench

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Georgia Chenevix-Trench

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Anna deFazio

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Jeremy M Arnold

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Joshy George

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Anna V Tinker

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Lisa Simms

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Bianca Locandro

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Sian Fereday

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Nadia Traficante

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Peter Russell

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Raghwa Sharma

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Michael J Birrer

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AOCS Study Group

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David D L Bowtell

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publication date

1 November 2008

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Molecular Cancer Research

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11

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1678-1690

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13 May 2020

Single nucleotide polymorphism genotyping using allele-specific PCR and fluorescence melting curves

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In serous ovarian neoplasms the frequency of Ki-ras mutations correlates with their malignant potential

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SPRY2 is an inhibitor of the ras/extracellular signal-regulated kinase pathway in melanocytes and melanoma cells with wild-type BRAF but not with the V599E mutant

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1 reference

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Mutations of BRAF and KRAS precede the development of ovarian serous borderline tumors

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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Identifiers

10.1158/1541-7786.MCR-08-0193

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Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=PMCID:PMC6953412&resulttype=core&format=json

13 May 2020

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Europe PubMed Central

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13 May 2020

1 reference

Europe PubMed Central

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13 May 2020

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