Proteolysis by presenilins and the renaissance of tau. (Q53229481)

12 May 2018

title

Proteolysis by presenilins and the renaissance of tau. (English)

1 reference

12 May 2018

1 reference

Haass C

1

1 reference

12 May 2018

Mandelkow E

series ordinal

2

1 reference

12 May 2018

publication date

1 June 1999

1 reference

12 May 2018

published in

Trends in Cell Biology

1 reference

12 May 2018

volume

9

1 reference

12 May 2018

issue

6

1 reference

12 May 2018

page(s)

241-244

1 reference

12 May 2018

The cell biology of beta-amyloid precursor protein and presenilin in Alzheimer's disease

cites work

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Tau in Alzheimer's disease.

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Two transmembrane aspartates in presenilin-1 required for presenilin endoproteolysis and gamma-secretase activity

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Deficiency of presenilin-1 inhibits the normal cleavage of amyloid precursor protein

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Effects of PS1 deficiency on membrane protein trafficking in neurons.

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Skeletal and CNS defects in Presenilin-1-deficient mice

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Presenilin 1 is required for Notch1 and DII1 expression in the paraxial mesoderm

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Facilitation of lin-12-mediated signalling by sel-12, a Caenorhabditis elegans S182 Alzheimer's disease gene

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Caspase-mediated cleavage is not required for the activity of presenilins in amyloidogenesis and NOTCH signaling.

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Nuclear access and action of notch in vivo

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Notch-1 signalling requires ligand-induced proteolytic release of intracellular domain

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Formation of stable complexes between two Alzheimer's disease gene products: presenilin-2 and beta-amyloid precursor protein

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Interaction between amyloid precursor protein and presenilins in mammalian cells: implications for the pathogenesis of Alzheimer disease

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Expression of Alzheimer's disease-associated presenilin-1 is controlled by proteolytic degradation and complex formation

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Stable association of presenilin derivatives and absence of presenilin interactions with APP.

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Protein topology of presenilin 1

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Neuropathological stageing of Alzheimer-related changes

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Frequency of stages of Alzheimer-related lesions in different age categories: concurrences and cautions.

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Neurofibrillary tangles but not senile plaques parallel duration and severity of Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Detection of tau proteins in normal and Alzheimer's disease cerebrospinal fluid with a sensitive sandwich enzyme-linked immunosorbent assay.

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Tau protein pathology in neurodegenerative diseases.

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Tau is a candidate gene for chromosome 17 frontotemporal dementia

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Mutation in the tau gene in familial multiple system tauopathy with presenile dementia

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Association of missense and 5'-splice-site mutations in tau with the inherited dementia FTDP-17

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Pathogenic implications of mutations in the tau gene in pallido-ponto-nigral degeneration and related neurodegenerative disorders linked to chromosome 17

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Tau proteins with FTDP-17 mutations have a reduced ability to promote microtubule assembly

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Mutation-specific functional impairments in distinct tau isoforms of hereditary FTDP-17.

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Overexpression of tau protein inhibits kinesin-dependent trafficking of vesicles, mitochondria, and endoplasmic reticulum: implications for Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Phosphorylation that detaches tau protein from microtubules (Ser262, Ser214) also protects it against aggregation into Alzheimer paired helical filaments

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

The development of cell processes induced by tau protein requires phosphorylation of serine 262 and 356 in the repeat domain and is inhibited by phosphorylation in the proline-rich domains

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Transgenic expression of the shortest human tau affects its compartmentalization and its phosphorylation as in the pretangle stage of Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Altered microtubule organization in small-calibre axons of mice lacking tau protein

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

A presenilin-1-dependent gamma-secretase-like protease mediates release of Notch intracellular domain

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Presenilin is required for activity and nuclear access of Notch in Drosophila

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

Neurogenic phenotypes and altered Notch processing in Drosophila Presenilin mutants.

1 reference

https://api.crossref.org/works/10.1016%2FS0962-8924%2899%2901576-7

7 January 2021

10.1016/S0962-8924(99)01576-7

Identifiers

DOI

1 reference

12 May 2018

PubMed ID

10408925

1 reference

12 May 2018