Absence of TMPRSS2:ERG fusions and PTEN losses in prostate cancer is associated with a favorable outcome (Q57790493)

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Anthony M Joshua

object named as

Anthony M Joshua

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author name string

Maisa Yoshimoto

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Isabela W Cunha

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Renata A Coudry

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Francisco P Fonseca

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Olga Ludkovski

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Maria Zielenska

https://scigraph.springernature.com/pub.10.1038/modpathol.2008.96

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publication date

23 May 2008

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published in

Modern Pathology

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volume

21

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issue

12

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page(s)

1451-1460

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exact match

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cites work

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Interphase FISH analysis of PTEN in histologic sections shows genomic deletions in 68% of primary prostate cancer and 23% of high-grade prostatic intra-epithelial neoplasias

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Three-color FISH analysis of TMPRSS2/ERG fusions in prostate cancer indicates that genomic microdeletion of chromosome 21 is associated with rearrangement

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mTOR inhibition reverses Akt-dependent prostate intraepithelial neoplasia through regulation of apoptotic and HIF-1-dependent pathways

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Akt promotes cell survival by phosphorylating and inhibiting a Forkhead transcription factor

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Increased AKT activity contributes to prostate cancer progression by dramatically accelerating prostate tumor growth and diminishing p27Kip1 expression

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Complete loss of PTEN expression as a possible early prognostic marker for prostate cancer metastasis

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FISH analysis of 107 prostate cancers shows that PTEN genomic deletion is associated with poor clinical outcome.

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TMPRSS2:ETV4 gene fusions define a third molecular subtype of prostate cancer

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TMPRSS2:ERG fusion-associated deletions provide insight into the heterogeneity of prostate cancer

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TMPRSS2-ERG gene fusion causing ERG overexpression precedes chromosome copy number changes in prostate carcinomas and paired HGPIN lesions

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Duplication of the fusion of TMPRSS2 to ERG sequences identifies fatal human prostate cancer

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Loss of PTEN is associated with progression to androgen independence

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ETS-TMPRSS2 fusion gene products in prostate cancer.

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Gene expression profiling predicts clinical outcome of prostate cancer

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