E6 variants of human papillomavirus 18 differentially modulate the protein kinase B/phosphatidylinositol 3-kinase (akt/PI3K) signaling pathway (Q39917232)

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English	E6 variants of human papillomavirus 18 differentially modulate the protein kinase B/phosphatidylinositol 3-kinase (akt/PI3K) signaling pathway	scientific article

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

15 September 2017

E6 variants of human papillomavirus 18 differentially modulate the protein kinase B/phosphatidylinositol 3-kinase (akt/PI3K) signaling pathway (English)

1 reference

Europe PubMed Central

PubMed publication ID

15 September 2017

author name string

Adriana Contreras-Paredes

1

1 reference

Europe PubMed Central

PubMed publication ID

15 September 2017

Erick De la Cruz-Hernández

2

1 reference

Europe PubMed Central

PubMed publication ID

15 September 2017

Imelda Martínez-Ramírez

3

1 reference

Europe PubMed Central

PubMed publication ID

15 September 2017

Alfonso Dueñas-González

4

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Europe PubMed Central

PubMed publication ID

15 September 2017

Marcela Lizano

5

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Europe PubMed Central

PubMed publication ID

15 September 2017

language of work or name

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publication date

12 November 2008

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Europe PubMed Central

PubMed publication ID

15 September 2017

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Europe PubMed Central

PubMed publication ID

15 September 2017

383

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Europe PubMed Central

PubMed publication ID

15 September 2017

1

1 reference

Europe PubMed Central

PubMed publication ID

15 September 2017

78-85

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Europe PubMed Central

PubMed publication ID

15 September 2017

Molecular analysis of the PI3K-AKT pathway in uterine cervical neoplasia: frequent PIK3CA amplification and AKT phosphorylation

1 reference

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The causal relation between human papillomavirus and cervical cancer

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Melanocortin-3 receptor activates MAP kinase via PI3 kinase

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Human papillomavirus type 16 E6 amino acid 83 variants enhance E6-mediated MAPK signaling and differentially regulate tumorigenesis by notch signaling and oncogenic Ras.

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Differential splicing of E6 within human papillomavirus type 18 variants and functional consequences

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RNA interference in mammalian cells using siRNAs synthesized with T7 RNA polymerase

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PI 3-kinase, Akt and cell survival

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A Novel Mitogenic Signaling Pathway of Bradykinin in the Human Colon Carcinoma Cell Line SW-480 Involves Sequential Activation of a Gq/11 Protein, Phosphatidylinositol 3-Kinase , and Protein Kinase C

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Degradation of hDlg and MAGIs by human papillomavirus E6 is E6-AP-independent

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Methylation-specific PCR: a novel PCR assay for methylation status of CpG islands

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Human papillomavirus type 16 variants and risk of cervical cancer

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The APC-hDLG complex negatively regulates cell cycle progression from the G0/G1 to S phase

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Human papillomavirus type 16 E6 protein interacts with cystic fibrosis transmembrane regulator-associated ligand and promotes E6-associated protein-mediated ubiquitination and proteasomal degradation

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Id-1 is induced in MDCK epithelial cells by activated Erk/MAPK pathway in response to expression of the Snail and E47 transcription factors.

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Variants of the long control region and the E6 oncogene in European human papillomavirus type 16 isolates: implications for cervical disease

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Genetic alterations and aberrant expression of genes related to the phosphatidyl-inositol-3'-kinase/protein kinase B (Akt) signal transduction pathway in glioblastomas.

1 reference

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Gi-mediated activation of the Ras/MAP kinase pathway involves a 100 kDa tyrosine-phosphorylated Grb2 SH3 binding protein, but not Src nor Shc

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Human homolog of disc-large is required for adherens junction assembly and differentiation of human intestinal epithelial cells

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Binding of human virus oncoproteins to hDlg/SAP97, a mammalian homolog of the Drosophila discs large tumor suppressor protein

1 reference

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Expression of PTEN in the progression of cervical neoplasia and its relation to tumor behavior and angiogenesis in invasive squamous cell carcinoma

1 reference

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PTEN, a putative protein tyrosine phosphatase gene mutated in human brain, breast, and prostate cancer

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Differential expression of human Dlg in cervical intraepithelial neoplasias

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Association between human papillomavirus type 18 variants and histopathology of cervical cancer.

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PIK3CA as an oncogene in cervical cancer

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Human papillomavirus type 16 E6 promotes retinoblastoma protein phosphorylation and cell cycle progression.

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The human papillomavirus E6 protein and its contribution to malignant progression

1 reference

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HPV E6 specifically targets different cellular pools of its PDZ domain-containing tumour suppressor substrates for proteasome-mediated degradation

1 reference

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Activated Notch1 inhibits p53-induced apoptosis and sustains transformation by human papillomavirus type 16 E6 and E7 oncogenes through a PI3K-PKB/Akt-dependent pathway

1 reference

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Frequent genetic and biochemical alterations of the PI 3-K/AKT/PTEN pathway in head and neck squamous cell carcinoma

1 reference

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Activation of the protein kinase B pathway by the HPV-16 E7 oncoprotein occurs through a mechanism involving interaction with PP2A

1 reference

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Phosphatidylinositol 3-kinases in tumor progression

1 reference

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Mutation analysis of the putative tumor suppressor gene PTEN/MMAC1 in cervical cancer

1 reference

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PTEN is a negative regulator of STAT3 activation in human papillomavirus-infected cells

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HPV E6 and MAGUK protein interactions: determination of the molecular basis for specific protein recognition and degradation

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Biological role of phosphatase PTEN in cancer and tissue injury healing

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Binding of PTEN to specific PDZ domains contributes to PTEN protein stability and phosphorylation by microtubule-associated serine/threonine kinases

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Induction of cell death in human papillomavirus 18-positive cervical cancer cells by E6 siRNA.

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Differential DNA methylation profiles in gynecological cancers and correlation with clinico-pathological data

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Structures of a human papillomavirus (HPV) E6 polypeptide bound to MAGUK proteins: mechanisms of targeting tumor suppressors by a high-risk HPV oncoprotein

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Considerations when analyzing the methylation status of PTEN tumor suppressor gene

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Natural variants of the human papillomavirus type 16 E6 protein differ in their abilities to alter keratinocyte differentiation and to induce p53 degradation.

1 reference

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10.1016/J.VIROL.2008.09.040

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Europe PubMed Central

PubMed publication ID

15 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

15 September 2017

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