Upregulation of BiP and CHOP by the unfolded-protein response is independent of presenilin expression. (Q40833574)

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3 November 2019

title

Upregulation of BiP and CHOP by the unfolded-protein response is independent of presenilin expression (English)

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3 November 2019

9

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3 November 2019

Amy S Lee

8

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3 November 2019

author name string

Sato N

1

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3 November 2019

Urano F

2

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3 November 2019

Yoon Leem J

3

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3 November 2019

Kim SH

4

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3 November 2019

Li M

5

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3 November 2019

Donoviel D

6

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Bernstein A

7

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Veselits ML

10

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Sisodia SS

11

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Thinakaran G

12

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3 November 2019

publication date

1 December 2000

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3 November 2019

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3 November 2019

volume

2

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issue

12

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3 November 2019

page(s)

863-870

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https://scigraph.springernature.com/pub.10.1038/35046500

3 November 2019

exact match

0 references

cites work

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A presenilin-1-dependent gamma-secretase-like protease mediates release of Notch intracellular domain

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Secreted amyloid beta-protein similar to that in the senile plaques of Alzheimer's disease is increased in vivo by the presenilin 1 and 2 and APP mutations linked to familial Alzheimer's disease

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Familial Alzheimer's disease-linked presenilin 1 variants elevate Abeta1-42/1-40 ratio in vitro and in vivo

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The presenilin 2 mutation (N141I) linked to familial Alzheimer disease (Volga German families) increases the secretion of amyloid beta protein ending at the 42nd (or 43rd) residue

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A role for presenilin-1 in nuclear accumulation of Ire1 fragments and induction of the mammalian unfolded protein response

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Presenilin-1 mutations downregulate the signalling pathway of the unfolded-protein response

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Stress signaling from the lumen of the endoplasmic reticulum: coordination of gene transcriptional and translational controls

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Signal transduction from the endoplasmic reticulum to the cell nucleus

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Oligomerization and phosphorylation of the Ire1p kinase during intracellular signaling from the endoplasmic reticulum to the nucleus

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The transmembrane kinase Ire1p is a site-specific endonuclease that initiates mRNA splicing in the unfolded protein response

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7 January 2021

The unfolded protein response: an intracellular signalling pathway with many surprising features

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7 January 2021

Dynamic interaction of BiP and ER stress transducers in the unfolded-protein response

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Signals from the stressed endoplasmic reticulum induce C/EBP-homologous protein (CHOP/GADD153).

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Perk is essential for translational regulation and cell survival during the unfolded protein response

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7 January 2021

Identification and characterization of pancreatic eukaryotic initiation factor 2 alpha-subunit kinase, PEK, involved in translational control

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Protein translation and folding are coupled by an endoplasmic-reticulum-resident kinase

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Evidence that levels of presenilins (PS1 and PS2) are coordinately regulated by competition for limiting cellular factors.

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An increased percentage of long amyloid beta protein secreted by familial amyloid beta protein precursor (beta APP717) mutants.

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7 January 2021

Processing of presenilin 1 in brains of patients with Alzheimer's disease and controls.

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7 January 2021

A vector for expressing foreign genes in the brains and hearts of transgenic mice

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Accelerated amyloid deposition in the brains of transgenic mice coexpressing mutant presenilin 1 and amyloid precursor proteins.

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7 January 2021

Presenilins, the endoplasmic reticulum, and neuronal apoptosis in Alzheimer's disease.

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7 January 2021

Presenilin 1 is required for Notch1 and DII1 expression in the paraxial mesoderm

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7 January 2021

Mice lacking both presenilin genes exhibit early embryonic patterning defects

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7 January 2021

Metabolism of the "Swedish" amyloid precursor protein variant in neuro2a (N2a) cells. Evidence that cleavage at the "beta-secretase" site occurs in the golgi apparatus

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7 January 2021

The nonconserved hydrophilic loop domain of presenilin (PS) is not required for PS endoproteolysis or enhanced abeta 42 production mediated by familial early onset Alzheimer's disease-linked PS variants.

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7 January 2021

Molecular genetic analysis of familial early-onset Alzheimer's disease linked to chromosome 14q24.3.

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Stable association of presenilin derivatives and absence of presenilin interactions with APP.

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7 January 2021

ATF6 as a transcription activator of the endoplasmic reticulum stress element: thapsigargin stress-induced changes and synergistic interactions with NF-Y and YY1

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7 January 2021

Identifiers

DOI

10.1038/35046500

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:11146649%20AND%20SRC:MED&resulttype=core&format=json

3 November 2019

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