Nifedipine protects INS-1 β-cell from high glucose-induced ER stress and apoptosis (Q35600131)

9 August 2017

Nifedipine protects INS-1 β-cell from high glucose-induced ER stress and apoptosis (English)

1 reference

9 August 2017

1 reference

Yao Wang

1

1 reference

9 August 2017

Lu Gao

2

1 reference

9 August 2017

Yuan Li

3

1 reference

9 August 2017

Hong Chen

4

1 reference

9 August 2017

Zilin Sun

5

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9 August 2017

language of work or name

English

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publication date

7 November 2011

1 reference

International Journal of Molecular Sciences

9 August 2017

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9 August 2017

12

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9 August 2017

11

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9 August 2017

7569-7580

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Creative Commons Attribution 4.0 International

9 August 2017

start time

7 November 2011

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April 2022 Public Data File from Crossref

copyright status

copyrighted

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cites work

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Sulfonylureas uncouple glucose-dependence for GPR40-mediated enhancement of insulin secretion from INS-1E cells

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Suppression of p38 MAPK and JNK via Akt-mediated inhibition of apoptosis signal-regulating kinase 1 constitutes a core component of the beta-cell pro-survival effects of glucose-dependent insulinotropic polypeptide

12 July 2018

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12 July 2018

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12 July 2018

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12 July 2018

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12 July 2018

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12 July 2018

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12 July 2018

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12 July 2018

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12 July 2018

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Nifedipine. A review of its pharmacodynamic and pharmacokinetic properties, and therapeutic efficacy, in ischaemic heart disease, hypertension and related cardiovascular disorders

12 July 2018

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12 July 2018

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Glucose- and interleukin-1beta-induced beta-cell apoptosis requires Ca2+ influx and extracellular signal-regulated kinase (ERK) 1/2 activation and is prevented by a sulfonylurea receptor 1/inwardly rectifying K+ channel 6.2 (SUR/Kir6.2) selective po

12 July 2018

1 reference

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Requirements of calcium fluxes and ERK kinase activation for glucose- and interleukin-1beta-induced beta-cell apoptosis

21 January 2018

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Glucose induces beta-cell apoptosis via upregulation of the Fas receptor in human islets

21 January 2018

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Prolonged exposure to free fatty acids has cytostatic and pro-apoptotic effects on human pancreatic islets: evidence that beta-cell death is caspase mediated, partially dependent on ceramide pathway, and Bcl-2 regulated.

21 January 2018

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Glucose-induced endoplasmic reticulum stress is independent of oxidative stress: A mechanistic explanation for the failure of antioxidant therapy in diabetes.

21 January 2018

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Glucose and palmitic acid induce degeneration of myofibrils and modulate apoptosis in rat adult cardiomyocytes

21 January 2018

1 reference

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IL-1beta caused pancreatic beta-cells apoptosis is mediated in part by endoplasmic reticulum stress via the induction of endoplasmic reticulum Ca2+ release through the c-Jun N-terminal kinase pathway

21 January 2018

1 reference

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Bid is cleaved by calpain to an active fragment in vitro and during myocardial ischemia/reperfusion

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Comparison of metabolic oscillations from mouse pancreatic beta cells and islets

21 January 2018

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Activation of liver X receptors inhibits pancreatic islet beta cell proliferation through cell cycle arrest

21 January 2018

1 reference

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Nifedipine prevents apoptosis of endothelial cells induced by oxidized low-density lipoproteins

21 January 2018

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High glucose-induced apoptosis through store-operated calcium entry and calcineurin in human umbilical vein endothelial cells

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Ca2+ channel blockers verapamil and nifedipine inhibit apoptosis induced by 25-hydroxycholesterol in human aortic smooth muscle cells

21 January 2018

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β-Amyloid-induced calcium influx induces apoptosis in culture by oxidative stress rather than tau phosphorylation

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Sensitivity of pancreatic beta cell to calcium channel blockers. An electrophysiologic study of verapamil and nifedipine

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Oscillations of secretion driven by oscillations of cytoplasmic Ca2+ as evidences in single pancreatic islets

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Exocytosis elicited by action potentials and voltage-clamp calcium currents in individual mouse pancreatic B-cells

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Identifiers

DOI

10.3390/IJMS12117569

1 reference

9 August 2017

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9 August 2017

1 reference